Evidence map›Paper›PMID 38649860›Full record

ArticleBMC cancer2024

m

Yingmin Wu, Lian Li, Long Wang, Shenjie Zhang, Zhirui Zeng, Jieyu Lu, Zhi Wang, Yewei Zhang, Shilong Zhang, Haiyang Li and 1 more

Abstract read
In one paragraph

Article in BMC cancer, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed.

  1. Review
  2. mCell death & disease · 2026
    Article
  3. Article
  4. Review
  5. Article
  6. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors.

Yingmin Wu *Department of Physiology, School of Basic Medical Sciences, Guizhou Medical University, 561113, Guiyang, China. wuyingmin1990@126.com.
Lian Li *Department of Physiology, School of Basic Medical Sciences, Guizhou Medical University, 561113, Guiyang, China.
Long Wang *Department of Physiology, School of Basic Medical Sciences, Guizhou Medical University, 561113, Guiyang, China.
Shenjie ZhangDepartment of Surgery, Affiliated Hospital of Guizhou Medical University, 550004, Guiyang, China.
Zhirui ZengDepartment of Physiology, School of Basic Medical Sciences, Guizhou Medical University, 561113, Guiyang, China.
Jieyu LuDepartment of Physiology, School of Basic Medical Sciences, Guizhou Medical University, 561113, Guiyang, China.
Zhi WangDepartment of Surgery, Affiliated Hospital of Guizhou Medical University, 550004, Guiyang, China.
Yewei ZhangDepartment of Surgery, Affiliated Hospital of Guizhou Medical University, 550004, Guiyang, China.
Shilong ZhangDepartment of Surgery, Affiliated Hospital of Guizhou Medical University, 550004, Guiyang, China.
Haiyang LiDepartment of Surgery, Affiliated Hospital of Guizhou Medical University, 550004, Guiyang, China. lihaiyang@gmc.edu.cn.
Tengxiang ChenDepartment of Physiology, School of Basic Medical Sciences, Guizhou Medical University, 561113, Guiyang, China. txch@gmc.edu.cn.

Funding

Guizhou Medical University Cultivation project of National Natural Science Foundation 20NSP009Guizhou Provincial Department of Education for Higher Education Scientific Research Foundation QJJ[2022]191Guizhou Provincial Health Commission Science and Technology Foundation gzwkj2022-254The China Postdoctoral Science Foundation 2021M700971The Excellent Young Talents Plan of Guizhou Medical University [2022]109The Guizhou Provincial Science and Technology Projects ZK[2022]390The National Natural Science Foundation of China 82103296 and 82260606
6 · The paper itself

Abstract

backgroundN1-methyladenosine (m

methodsAn integrated analysis of the TCGA-LIHC database was performed to explore the gene signatures and clinical relevance of 10 m

resultsThe risk model, constructed from five DEGs (CDK5R2, TRIM36, DCAF8L, CYP26B, and PAGE1), exhibited significant prognostic value in predicting survival rates among individuals with HCC. Moreover, HCC tissues showed decreased levels of m

conclusionsThe risk model, comprising m

Indexed as

AutophagyCarcinoma, HepatocellularGene Expression Regulation, NeoplasticRNA MethylationAdenosineBiomarkers, TumorCell Line, TumorCell ProliferationDNA MethylationFemaleGene Expression ProfilingHumansLiver NeoplasmsMalePrognosisAdenosineBiomarkers, TumorN-methyladenosineAutophagyHepatocellular carcinoma (HCC)m1A regulatorsN1-methyladenosine (m1A)Prognosis

Identifiers

PMID38649860
PMCPMC11034060

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.