Evidence map›Paper›PMID 38647760›Full record

ArticleBioresources and bioprocessing2022

Toxicity and inhibition mechanism of gallic acid on physiology and fermentation performance of Escherichia coli.

Lina Liu, Xiaolong Ma, Muhammad Bilal, Linlin Wei, Shijie Tang, Hongzhen Luo, Yuping Zhao, Zhaoyu Wang, Xuguo Duan

Open access · diamondAbstract read
In one paragraph

Article in Bioresources and bioprocessing, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
1.3field-weighted citation impact, top 14% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed, 14 citations in OpenAlex.

  1. Article
  2. Infant-derivedFrontiers in nutrition · 2026
    Article
  3. Article
  4. Review
  5. Review
  6. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 2 institutions in 1 country.

Lina LiuSchool of Life Science and Food Engineering, Huaiyin Institute of Technology, Huaian, 223003, China. linaliu@hyit.edu.cn.ORCID http://orcid.org/0000-0003-2970-9788
Xiaolong MaSchool of Life Science and Food Engineering, Huaiyin Institute of Technology, Huaian, 223003, China.
Muhammad BilalSchool of Life Science and Food Engineering, Huaiyin Institute of Technology, Huaian, 223003, China.
Linlin WeiSchool of Life Science and Food Engineering, Huaiyin Institute of Technology, Huaian, 223003, China.
Shijie TangSchool of Life Science and Food Engineering, Huaiyin Institute of Technology, Huaian, 223003, China.
Hongzhen LuoSchool of Life Science and Food Engineering, Huaiyin Institute of Technology, Huaian, 223003, China.
Yuping ZhaoSchool of Life Science and Food Engineering, Huaiyin Institute of Technology, Huaian, 223003, China.
Zhaoyu WangSchool of Life Science and Food Engineering, Huaiyin Institute of Technology, Huaian, 223003, China.
Xuguo DuanCollege of Light Industry and Food Engineering, Nanjing Forestry University, Nanjing, 210037, Jiangsu, China.
Huaiyin Institute of Technology · CNNanjing Forestry University · CN

Funding

Innovative Research Group Project of the National Natural Science Foundation of China 21808075Natural Science Foundation of Jiangsu Province BK20170459
6 · The paper itself

Abstract

Gallic acid is a natural phenolic acid that has a stress inhibition effect on Escherichia coli. This study by integrates fermentation characteristics and transcriptional analyses to elucidate the physiological mechanism of E. coli 3110 response to gallic acid. Compared with the control (without stress), the cell growth was severely retarded, and irregular cell morphology appeared in the case of high levels of gallic acid stress. The glucose consumption of E. coli was reduced successively with the increase of gallic acid content in the fermentation medium. After 20 h of gallic acid stress, cofactor levels (ATP, NAD

Indexed as

Escherichia coliFermentation characteristicsGallic acidPhysiological mechanismTranscriptional analysis

Identifiers

PMID38647760
PMCPMC10992115
OpenAlexW4286715757

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.