Evidence map›Paper›PMID 38601492›Full record

ArticleFrontiers in public health2024

No causal association between pneumoconiosis and three inflammatory immune diseases: a Mendelian randomization study.

Yu-Jie Du, Zhang-Wei Lu, Kai-Di Li, Yi-Yu Wang, Hong Wu, Rong-Gui Huang, Xue Jin, Yi-Yuan Wang, Jing Wang, An-Yi Geng and 1 more

Open access · goldAbstract read
In one paragraph

Article in Frontiers in public health, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed, 1 pooled it
0.2field-weighted citation impact, top 46% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 1 synthesis or guideline pooled it, 1 citations in OpenAlex.

  1. Pooled it
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors at 1 institution in 1 country.

Yu-Jie Du *Department of Epidemiology and Biostatistics, School of Public Health, Anhui Medical University, Hefei, China.
Zhang-Wei Lu *Department of Epidemiology and Biostatistics, School of Public Health, Anhui Medical University, Hefei, China.
Kai-Di LiDepartment of Epidemiology and Biostatistics, School of Public Health, Anhui Medical University, Hefei, China.
Yi-Yu WangDepartment of Epidemiology and Biostatistics, School of Public Health, Anhui Medical University, Hefei, China.
Hong WuDepartment of Epidemiology and Biostatistics, School of Public Health, Anhui Medical University, Hefei, China.
Rong-Gui HuangDepartment of Epidemiology and Biostatistics, School of Public Health, Anhui Medical University, Hefei, China.
Xue JinDepartment of Epidemiology and Biostatistics, School of Public Health, Anhui Medical University, Hefei, China.
Yi-Yuan WangDepartment of Epidemiology and Biostatistics, School of Public Health, Anhui Medical University, Hefei, China.
Jing WangDepartment of Epidemiology and Biostatistics, School of Public Health, Anhui Medical University, Hefei, China.
An-Yi GengDepartment of Epidemiology and Biostatistics, School of Public Health, Anhui Medical University, Hefei, China.
Bao-Zhu LiDepartment of Epidemiology and Biostatistics, School of Public Health, Anhui Medical University, Hefei, China.
Anhui Medical University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Objectives: To investigate the causal relationships between pneumoconiosis and rheumatoid arthritis (RA), systemic lupus erythematosus (SLE), and gout. Methods: The random-effects inverse variance weighted (IVW) approach was utilized to explore the causal effects of the instrumental variables (IVs). Sensitivity analyses using the MR-Egger and weighted median (WM) methods were did to investigate horizontal pleiotropy. A leave-one-out analysis was used to avoid the bias resulting from single-nucleotide polymorphisms (SNPs). Results: There was no causal association between pneumoconiosis and SLE, RA or gout in the European population [OR = 1.01, 95% CI: 0.94-1.10, Conclusion: Our study suggests that pneumoconiosis may have no causal relationship with the three inflammatory immune diseases.

Indexed as

GoutImmune System DiseasesLupus Erythematosus, SystemicPneumoconiosisHumansMendelian Randomization Analysiscausal relationshipgoutinflammatory immune diseasesrheumatoid arthritissystemic lupus erythematosus

Identifiers

PMID38601492
PMCPMC11004292
OpenAlexW4393231430

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.