Evidence map›Paper›PMID 38575214›Full record

ReviewImmunology and allergy clinics of North America2024

Epidemiologic and Clinical Clues to the Etiology of Eosinophilic Esophagitis.

Joy W Chang, Elizabeth T Jensen

Abstract readReview
In one paragraph

Review in Immunology and allergy clinics of North America, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed.

  1. Review
  2. Review
  3. The Dynamic Evolution of Eosinophilic Esophagitis.Diagnostics (Basel, Switzerland) · 2025
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Joy W ChangDivision of Gastroenterology, Department of Internal Medicine, 3912 Taubman Center, 1500 East Medical Center Drive, SPC 5362, Ann Arbor, MI 48109, USA. Electronic address: CHJOY@med.umich.edu.
Elizabeth T JensenDepartment of Epidemiology and Prevention, Wake Forest University School of Medicine, 475 Vine Street, Winston-Salem, NC 27101, USA.

Funding

Epidemiologic identification and mechanistic investigation of early life environmental risk factors for eosinophilic esophagitisR01ES031940 · NIEHS · UNIV OF NORTH CAROLINA CHAPEL HILL · PI DELLON, EVAN SAMUEL, JENSEN, ELIZABETH T · 2021 to 2025
$3.2M
Early life factors, gene-environment interaction and eosinophilic esophagitisR01AI139126 · NIAID · WAKE FOREST UNIVERSITY HEALTH SCIENCES · PI JENSEN, ELIZABETH T · 2018 to 2023
$1.7M
Developing Patient-Centered Decision Support for Eosinophilic EsophagitisK23DK129784 · NIDDK · UNIVERSITY OF MICHIGAN AT ANN ARBOR · PI Joy Weiling CHANG · 2022 to 2026
$1.1M
NIAID NIH HHS R01 AI139126NIDDK NIH HHS K23 DK129784NIEHS NIH HHS R01 ES031940
6 · The paper itself

Abstract

Despite the rising prevalence and incidence of eosinophilic esophagitis (EoE), the etiology and pathophysiology remain unknown. Studies to date suggest that complex interactions between genetic and environmental risk factors result in the development and presentation of disease. Examining environmental factors both in the early life and later life exposures offers potential clues for the development of EoE, although challenges exist in making causal inferences due to diagnostic delay and access, ascertainment biases, and misclassification of cases. The authors review studies supporting early life factors as etiologic factors in the development of EoE.

Indexed as

Eosinophilic EsophagitisDelayed DiagnosisHumansIncidencePrevalenceRisk FactorsEarly lifeEnvironmental factorsEosinophilic esophagitisEpidemiology

Identifiers

PMID38575214
PMCPMC11003716

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.