Evidence map›Paper›PMID 38558633›Full record

ArticleCureus2024

A Case of C3 Nephritis With a Rare Variant of the CFHR5 Gene.

Hisashi Kamido, Shinya Yamamoto, Hideki Yokoi, Masashi Mizuno, Motoko Yanagita

Open access · diamondAbstract readCase Reports
PubMed Full text at PMC Publisher
In one paragraph

Article in Cureus, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact, top 97% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed, 0 citations in OpenAlex.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 4 institutions in 1 country.

Hisashi KamidoNephrology, Graduate School of Medicine, Kyoto University, Kyoto, JPN.
Shinya YamamotoNephrology, Graduate School of Medicine, Kyoto University, Kyoto, JPN.
Hideki YokoiNephrology, Graduate School of Medicine, Kyoto University, Kyoto, JPN.
Masashi MizunoNephrology, Nagoya University, Nagoya, JPN.
Motoko YanagitaNephrology, Graduate School of Medicine, Kyoto University, Kyoto, JPN.
Nagoya University Hospital · JPInternational Institute for Advanced Studies · JPKyoto University · JPNagoya University · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

C3 nephropathy is a renal disease caused by the aberrant activation of the alternative complement pathway. The long-term renal prognosis of C3 nephropathy is generally poor, and elucidation of its pathogenesis is clinically important. Genetic abnormalities within complement genes, encompassing autoantibodies targeting complement components and complement factor H-related proteins (CFHRs), can lead to abnormal complement activation.

Indexed as

c3 nephritisc3 nephropathycfhr5complement factor h related proteinp453s

Identifiers

PMID38558633
PMCPMC10978818
OpenAlexW4392235495

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.