Evidence map›Paper›PMID 38557722›Full record

ArticleThe Journal of experimental medicine2024

Neutrophil IL-26 fuels autoinflammation.

Krisztina Futosi, Attila Mócsai

Open access · bronzeAbstract read
In one paragraph

Article in The Journal of experimental medicine, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
0.2field-weighted citation impact, top 46% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 1 citations in OpenAlex.

  1. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 1 institution in 1 country.

Krisztina FutosiDepartment of Physiology, Semmelweis University School of Medicine, Budapest, Hungary.ORCID 0000-0002-1661-4635
Attila MócsaiDepartment of Physiology, Semmelweis University School of Medicine, Budapest, Hungary.ORCID 0000-0002-0512-1157
Semmelweis University · HU

Funding

Hungarian National Research, Development, and Innovation Office KKP-129954HUN-REN Hungarian Research Network 0207007
6 · The paper itself

Abstract

Pustular psoriasis is an inflammatory skin disease with features of neutrophil-mediated sterile autoinflammation. In this issue of JEM, Baldo et al. (https://doi.org/10.1084/jem.20231464) show that this autoinflammation is driven by a vicious cycle through neutrophil-derived IL-26.

Indexed as

NeutrophilsPsoriasisHumansSignal Transduction

Identifiers

PMID38557722
PMCPMC10983689
OpenAlexW4393377025

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-SA
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.