Evidence map›Paper›PMID 38548269›Full record

ArticleThe American journal of pathology2024

Enhanced Optic Nerve Expansion and Altered Ultrastructure of Elastic Fibers Induced by Lysyl Oxidase Inhibition in a Mouse Model of Marfan Syndrome.

Hang-Jing Wu, Evan Krystofiak, John Kuchtey, Rachel W Kuchtey

Open access · hybridAbstract read
In one paragraph

Article in The American journal of pathology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
0.7field-weighted citation impact, top 32% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 3 citations in OpenAlex.

  1. Article
  2. In Search of Mouse Models for Exfoliation Syndrome.American journal of ophthalmology · 2024
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 1 country.

Hang-Jing WuVanderbilt Eye Institute, Vanderbilt University Medical Center, Nashville, Tennessee.
Evan KrystofiakCell Imaging Shared Resource, Vanderbilt University, Nashville, Tennessee.
John KuchteyVanderbilt Eye Institute, Vanderbilt University Medical Center, Nashville, Tennessee.
Rachel W KuchteyVanderbilt Eye Institute, Vanderbilt University Medical Center, Nashville, Tennessee; Department of Molecular Physiology and Biophysics, Vanderbilt University, Nashville, Tennessee. Electronic address: rachel.w.kuchtey@vumc.org.
Vanderbilt University Medical Center · USVanderbilt University · US

Funding

Tumor Immunology and Microenvironment Research ProgramP30CA068485 · NCI · VANDERBILT UNIVERSITY MEDICAL CENTER · PI Ben Ho Park · 1995 to 2026
$172.8M
Translational Analysis CoreP30DK058404 · NIDDK · VANDERBILT UNIVERSITY MEDICAL CENTER · PI RICHARD M. PEEK · 2002 to 2026
$29.9M
Vanderbilt Diabetes Research CenterP30DK020593 · NIDDK · VANDERBILT UNIVERSITY MEDICAL CENTER · PI ALVIN C POWERS · 2012 to 2026
$29.3M
Shop Module CoreP30EY008126 · NEI · VANDERBILT UNIVERSITY MEDICAL CENTER · PI David J. Calkins · 1989 to 2026
$19.6M
Vanderbilt Mouse Metabolic Physiology CenterU24DK059637 · NIDDK · VANDERBILT UNIVERSITY · PI WASSERMAN, DAVID H · 2001 to 2015
$14.9M
Targeting Tissue Biomechanics for Treatment of GlaucomaR01EY020894 · NEI · VANDERBILT UNIVERSITY MEDICAL CENTER · PI KUCHTEY, RACHEL W · 2010 to 2025
$6.4M
NCI NIH HHS P30 CA068485NEI NIH HHS P30 EY008126NEI NIH HHS R01 EY020894NIDDK NIH HHS P30 DK020593NIDDK NIH HHS P30 DK058404NIDDK NIH HHS U24 DK059637
6 · The paper itself

Abstract

Two major constituents of exfoliation material, fibrillin-1 and lysyl oxidase-like 1 (encoded by FBN1 and LOXL1), are implicated in exfoliation glaucoma, yet their individual contributions to ocular phenotype are minor. To test the hypothesis that a combination of FBN1 mutation and LOXL1 deficiency exacerbates ocular phenotypes, the pan-lysyl oxidase inhibitor β-aminopropionitrile (BAPN) was used to treat adult wild-type (WT) mice and mice heterozygous for a missense mutation in Fbn1 (Fbn1

Indexed as

AminopropionitrileDisease Models, AnimalFibrillin-1Marfan SyndromeOptic NerveProtein-Lysine 6-OxidaseAdipokinesAmino Acid OxidoreductasesAnimalsElastic TissueExtracellular Matrix ProteinsFibrillinsGlaucomaIntraocular PressureMiceMice, Inbred C57BLAdipokinesAmino Acid OxidoreductasesAminopropionitrileExtracellular Matrix ProteinsFbn1 protein, mouseFibrillin-1FibrillinsLoxl1 protein, mouseLox protein, mouseMicrofilament ProteinsProtein-Lysine 6-Oxidase

Identifiers

PMID38548269
PMCPMC11317902
OpenAlexW4393180816

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.