Evidence map›Paper›PMID 38546173›Full record

ReviewChannels (Austin, Tex.)2024

Ion channel-mediated mitochondrial volume regulation and its relationship with mitochondrial dynamics.

Yujia Zhuang, Wenting Jiang, Zhe Zhao, Wencui Li, Zhiqin Deng, Jianquan Liu

Open access · goldAbstract readReview
In one paragraph

Review in Channels (Austin, Tex.), 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
1.9field-weighted citation impact, top 15% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed, 8 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 1 institution in 1 country.

Yujia ZhuangHand and Foot Surgery Department, Shenzhen Second People's Hospital/the First Hospital Affiliated to Shenzhen University, Shenzhen, China.
Wenting JiangOperating room, Shenzhen Second People's Hospital/the First Hospital Affiliated to Shenzhen University, Shenzhen, China.
Zhe ZhaoHand and Foot Surgery Department, Shenzhen Second People's Hospital/the First Hospital Affiliated to Shenzhen University, Shenzhen, China.
Wencui LiHand and Foot Surgery Department, Shenzhen Second People's Hospital/the First Hospital Affiliated to Shenzhen University, Shenzhen, China.
Zhiqin DengHand and Foot Surgery Department, Shenzhen Second People's Hospital/the First Hospital Affiliated to Shenzhen University, Shenzhen, China.
Jianquan LiuHand and Foot Surgery Department, Shenzhen Second People's Hospital/the First Hospital Affiliated to Shenzhen University, Shenzhen, China.
Shenzhen Second People's Hospital · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The mitochondrion, one of the important cellular organelles, has the major function of generating adenosine triphosphate and plays an important role in maintaining cellular homeostasis, governing signal transduction, regulating membrane potential, controlling programmed cell death and modulating cell proliferation. The dynamic balance of mitochondrial volume is an important factor required for maintaining the structural integrity of the organelle and exerting corresponding functions. Changes in the mitochondrial volume are closely reflected in a series of biological functions and pathological changes. The mitochondrial volume is controlled by the osmotic balance between the cytoplasm and the mitochondrial matrix. Thus, any disruption in the influx of the main ion, potassium, into the cells can disturb the osmotic balance between the cytoplasm and the matrix, leading to water movement between these compartments and subsequent alterations in mitochondrial volume. Recent studies have shown that mitochondrial volume homeostasis is closely implicated in a variety of diseases. In this review, we provide an overview of the main influencing factors and research progress in the field of mitochondrial volume homeostasis.

Indexed as

Ion ChannelsMitochondrial DynamicsMitochondriaMitochondrial SizeSignal TransductionIon Channelscell volume regulationIon channelsmitochondrial

Identifiers

PMID38546173
PMCPMC10984129
OpenAlexW4393259660

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.