Evidence map›Paper›PMID 38543790›Full record

ArticleViruses2024

γδ T Cells Mediate a Requisite Portion of a Wound Healing Response Triggered by Cutaneous Poxvirus Infection.

Irene E Reider, Eugene Lin, Tracy E Krouse, Nikhil J Parekh, Amanda M Nelson, Christopher C Norbury

Open access · goldAbstract read
In one paragraph

Article in Viruses, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
1.6field-weighted citation impact, top 18% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 3 citations in OpenAlex.

  1. Article
  2. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 1 institution in 1 country.

Irene E ReiderDepartment of Microbiology & Immunology, Pennsylvania State University College of Medicine, Hershey, PA 17033, USA.
Eugene LinDepartment of Microbiology & Immunology, Pennsylvania State University College of Medicine, Hershey, PA 17033, USA.
Tracy E KrouseDepartment of Microbiology & Immunology, Pennsylvania State University College of Medicine, Hershey, PA 17033, USA.
Nikhil J ParekhDepartment of Microbiology & Immunology, Pennsylvania State University College of Medicine, Hershey, PA 17033, USA.
Amanda M NelsonDepartment of Dermatology, Pennsylvania State University College of Medicine, Hershey, PA 17033, USA.ORCID 0000-0001-6375-8706
Christopher C NorburyDepartment of Microbiology & Immunology, Pennsylvania State University College of Medicine, Hershey, PA 17033, USA.
Pennsylvania State University · US

Funding

Analysis of the mechanism of HCMV cytoplasmic envelopmentR01AI130156 · NIAID · PENNSYLVANIA STATE UNIV HERSHEY MED CTR · PI Christopher C Norbury · 2017 to 2026
$3.8M
How does Cytomegalovirus use interferon lambda for optimal spreadR21AI169180 · NIAID · PENNSYLVANIA STATE UNIV HERSHEY MED CTR · PI NORBURY, CHRISTOPHER C · 2022 to 2023
$446k
The Toponome of Virus Infected SkinR21AI121876 · NIAID · PENNSYLVANIA STATE UNIV HERSHEY MED CTR · PI NORBURY, CHRISTOPHER C · 2016 to 2017
$425k
Interferon-independent STAT1-mediated protective antiviral immunityR21AI133536 · NIAID · PENNSYLVANIA STATE UNIV HERSHEY MED CTR · PI NORBURY, CHRISTOPHER C · 2017 to 2018
$425k
The role of PTP1B in cell-to-cell spread of herpes simplex virus in vivoR21AI140014 · NIAID · PENNSYLVANIA STATE UNIV HERSHEY MED CTR · PI NORBURY, CHRISTOPHER C · 2018 to 2019
$422k
The effect of local virus infection upon cutaneous wound healing: the impact of virus-induced Type III IFNsR21AR077778 · NIAMS · PENNSYLVANIA STATE UNIV HERSHEY MED CTR · PI NORBURY, CHRISTOPHER C · 2021 to 2022
$390k
NIAID NIH HHS R01 AI130156NIAID NIH HHS R21 AI121876NIAID NIH HHS R21 AI133536NIAID NIH HHS R21 AI140014NIAID NIH HHS R21 AI169180NIAMS NIH HHS AR077778NIAMS NIH HHS R21 AR077778
6 · The paper itself

Abstract

Infection at barrier sites, e.g., skin, activates local immune defenses that limit pathogen spread, while preserving tissue integrity. Phenotypically distinct γδ T cell populations reside in skin, where they shape immunity to cutaneous infection prior to onset of an adaptive immune response by conventional αβ CD4

Indexed as

Poxviridae InfectionsSkinAdministration, CutaneousAnimalsHumansMiceMice, Inbred C57BLVaccinia virusWound Healingcutaneous infectionIL-10IL-22poxviruswound healingγδ T cell

Identifiers

PMID38543790
PMCPMC10975054
OpenAlexW4392654906

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.