Evidence map›Paper›PMID 38540440›Full record

ArticleGenes2024

Novel

Sayaka W Ozawa, Satomi Inomata, Yukiko Hata, Shinya Takarada, Mako Okabe, Hideyuki Nakaoka, Keijiro Ibuki, Naoki Nishida, Fukiko Ichida, Keiichi Hirono

Open access · goldAbstract readCase Reports
In one paragraph

Article in Genes, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
0.8field-weighted citation impact, top 27% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed, 2 citations in OpenAlex.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 2 institutions in 1 country.

Sayaka W OzawaDepartment of Pediatrics, Faculty of Medicine, University of Toyama, Toyama 930-0194, Japan.ORCID 0000-0002-9394-9746
Satomi InomataDepartment of Pediatrics, Faculty of Medicine, University of Toyama, Toyama 930-0194, Japan.
Yukiko HataLegal Medicine, Faculty of Medicine, University of Toyama, Toyama 930-0194, Japan.
Shinya TakaradaDepartment of Pediatrics, Faculty of Medicine, University of Toyama, Toyama 930-0194, Japan.
Mako OkabeDepartment of Pediatrics, Faculty of Medicine, University of Toyama, Toyama 930-0194, Japan.
Hideyuki NakaokaDepartment of Pediatrics, Faculty of Medicine, University of Toyama, Toyama 930-0194, Japan.
Keijiro IbukiDepartment of Pediatrics, Faculty of Medicine, University of Toyama, Toyama 930-0194, Japan.
Naoki NishidaLegal Medicine, Faculty of Medicine, University of Toyama, Toyama 930-0194, Japan.ORCID 0000-0002-6921-1768
Fukiko IchidaDepartment of Pediatrics, International University of Health and Welfare, Tokyo 107-0052, Japan.
Keiichi HironoDepartment of Pediatrics, Faculty of Medicine, University of Toyama, Toyama 930-0194, Japan.ORCID 0000-0002-4224-9053
University of Toyama · JPInternational University of Health and Welfare · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundLeft ventricular hypertrophy (LVH) is a well-recognized cardiac dysfunction in infants of mothers with gestational diabetes mellitus (GDM). Left ventricular noncompaction (LVNC) is a cardiomyopathy that is morphologically characterized by numerous prominent trabeculations and deep intertrabecular recesses on cardiovascular imaging. However, there have been no case reports on neonates of mothers with GDM showing LVH and LVNC. CASE PRESENTATION: A patient, with LVH of a mother with GDM, was delivered at 36 weeks of gestation. Prominent trabeculations in the LV, suggesting LVNC, instead of LVH, were apparent 1 week after birth. A heterozygous deletion variant in the

conclusionGenetic testing should be conducted to obtain an accurate outcome and medical care in a patient with LVH and subsequently prominent hypertrabeculation in the LV.

Indexed as

CardiomyopathiesDiabetes, GestationalHeart Defects, CongenitalCardiac MyosinsChild, PreschoolFemaleHumansHypertrophy, Left VentricularInfantInfant, NewbornMaleMothersMyosin Heavy ChainsPregnancyCardiac MyosinsMYH7 protein, humanMyosin Heavy Chainsgestational diabetes mellitusleft ventricular noncompactionMYH7 gene

Identifiers

PMID38540440
PMCPMC10969955
OpenAlexW4392990853

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.