Evidence map›Paper›PMID 38507154›Full record

ReviewCurrent cardiology reports2024

What Do We Know So Far About Ventricular Arrhythmias and Sudden Cardiac Death Prediction in the Mitral Valve Prolapse Population? Could Biomarkers Help Us Predict Their Occurrence?

D Dziadosz, L Daniłowicz-Szymanowicz, P Wejner-Mik, M Budnik, B Brzezińska, P Duchnowski, K Golińska-Grzybała, K Jaworski, I Jedliński, M Kamela and 16 more

Abstract readReview
In one paragraph

Review in Current cardiology reports, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

26 authors.

D Dziadosz1st Department of Cardiology, Faculty of Medicine, Medical University of Silesia, Katowice, Poland.
L Daniłowicz-SzymanowiczDepartment of Cardiology and Electrotherapy, Faculty of Medicine, Medical University of Gdańsk, Gdańsk, Poland.
P Wejner-Mik1st Department of Cardiology, Medical University of Lodz, Bieganski Hospital, Łódź, Poland.
M Budnik1st Chair and Department of Cardiology, Medical University of Warsaw, Central Clinical Hospital, 1a Banacha St, 02-97, Warsaw, Poland.
B BrzezińskaDepartment of Cardiology, T. Marciniak Hospital, Wrocław, Poland.
P DuchnowskiCardinal Wyszynski National Institute of Cardiology, 04-628, Warsaw, Poland.
K Golińska-GrzybałaDept of Coronary Disease and Heart Failure, Noninvasive Cardiovascular Laboratory, Medical College, Jagiellonian University, St. John Paul II Hospital, Cracow, Poland.
K JaworskiDepartment of Coronary Artery Disease and Cardiac Rehabilitation, National Institute of Cardiology, Warsaw, Poland.
I JedlińskiMedicor, Powstańców Wielkopolskich 4, 61-895, Poznań, Poland.
M KamelaDepartment of Cardiology, Hospital of the Ministry of Interior and Administration, Rzeszów, Poland.
J Kasprzak1st Department of Cardiology, Medical University of Lodz, Bieganski Hospital, Łódź, Poland.
M Kowalczyk-DomagałaPediatric Cardiology Department, The Children's Memorial Health Institute, Warsaw, Poland.
K KurnickaDepartment of Internal Medicine and Cardiology, Medical University of Warsaw, Infant Jesus Clinical Hospital, Lindleya str. 4, 02-005, Warsaw, Poland.
D Kustrzycka-KratochwilDepartment of Cardiology, Center for Heart Diseases, 4th Military Clinical Hospital, Weigla 5, 50-981, Wrocław, Poland.
K MickiewiczDepartment of Cardiology, Medical University of Bialystok, 15-276, Białystok, Poland.
O MożeńskaJO Medical Center, Quo Vadis 1/U6, 02-495, Warsaw, Poland.
Z Oko-SarnowskaDepartment of Cardiology, Poznań University of Medical Sciences, Wielkopolskie, 60-355, Poznań, Poland.
M PlewkaDepartment of Interventional Cardiology and Cardiac Arrhythmias, Military Medical Academy Memorial Teaching Hospital of the Medical University of Lodz, Łódź, Poland.
A PolewczykDepartment of Physiology, Pathophysiology and Clinical Immunology, Institute of Medical Sciences, Jan Kochanowski University, Żeromskiego 5, 25-369, Kielce, Poland.
B Uziębło-ŻyczkowskaDepartment of Cardiology and Internal Diseases, Military Institute of Medicine - National Research Institute, Warsaw, Poland.
K Wierzbowska-DrabikDepartment of Internal Medicine and Clinical Pharmacology, Medical University of Lodz, Łódź, Poland.
R Wachnicka-TrutyDepartment of Cardiology and Internal Diseases, Institute of Maritime and Tropical Medicine, Medical University of Gdańsk, Gdynia, Poland.
E Wołoszyn-HorákSecond Department of Cardiology. Specialist Hospital in Zabrze, Medical University of Silesia, Curie-Sklodowskiej str. 10, Zabrze, Poland.
P SzymańskiCenter of Clinical Cardiology, Central Clinical Hospital of the Ministry of Interior and Administration, Warsaw, Poland.
A GackowskiDept of Coronary Disease and Heart Failure, Noninvasive Cardiovascular Laboratory, Medical College, Jagiellonian University, St. John Paul II Hospital, Cracow, Poland.
K Mizia-Stec1st Department of Cardiology, Faculty of Medicine, Medical University of Silesia, Katowice, Poland. kmiziastec@gmail.com.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

purpose of the reviewTo summarize currently available data on the topic of mitral valve prolapse (MVP) and its correlation to the occurrence of atrial and ventricular arrhythmias. To assess the prognostic value of several diagnostic methods such as transthoracic echocardiography, transesophageal echocardiography, cardiac magnetic resonance, cardiac computed tomography, electrocardiography, and electrophysiology concerning arrhythmic episodes. To explore intra and extracellular biochemistry of the cardiovascular system and its biomarkers as diagnostic tools to predict rhythm disturbances in the MVP population. RECENT

findingsMVP is a common and mainly benign valvular disorder. It affects 2-3% of the general population. MVP is a heterogeneous and highly variable phenomenon with three structural phenotypes: myxomatous degeneration, fibroelastic deficiency, and forme fruste. Exercise intolerance, supraventricular tachycardia, and chest discomfort are the symptoms that are often paired with psychosomatic components. Though MVP is thought to be benign, the association between isolated MVP without mitral regurgitation (MR) or left ventricle dysfunction, with ventricular arrhythmia (VA) and sudden cardiac death (SCD) has been observed. The incidence of SCD in the MVP population is around 0.6% per year, which is 6 times higher than the occurrence of SCD in the general population. Often asymptomatic MVP population poses a challenge to screen for VA and prevent SCD. Therefore, it is crucial to carefully assess the risk of VA and SCD in patients with MVP with the use of various tools such as diagnostic imaging and biochemical and genetic screening.

Indexed as

BiomarkersDeath, Sudden, CardiacMitral Valve ProlapseArrhythmias, CardiacEchocardiographyElectrocardiographyHumansPrognosisRisk FactorsBiomarkersArrhythmic mitral valve prolapseMitral annulus disjunctionMitral valve prolapseVentricular arrhythmia

Identifiers

PMID38507154
PMCPMC11136782

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.