Evidence map›Paper›PMID 38505749›Full record

ArticleFrontiers in endocrinology2024

Double somatic mutations in

Kazutaka Nanba, Amy R Blinder, Aaron M Udager, Yuusuke Hirokawa, Takayoshi Miura, Hiroshi Okuno, Koki Moriyoshi, Yuto Yamazaki, Hironobu Sasano, Akihiro Yasoda and 3 more

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PubMed Full text at PMC Publisher
In one paragraph

Article in Frontiers in endocrinology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
2.1field-weighted citation impact, top 12% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed, 6 citations in OpenAlex.

  1. Article
  2. Article
  3. Review
  4. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors at 4 institutions in 2 countries.

Kazutaka NanbaDepartment of Endocrinology and Metabolism, National Hospital Organization Kyoto Medical Center, Kyoto, Japan.
Amy R BlinderDepartment of Molecular and Integrative Physiology, University of Michigan, Ann Arbor, MI, United States.
Aaron M UdagerDepartment of Pathology, University of Michigan, Ann Arbor, MI, United States.
Yuusuke HirokawaDepartment of Radiology, National Hospital Organization Kyoto Medical Center, Kyoto, Japan.
Takayoshi MiuraDepartment of Urology, National Hospital Organization Kyoto Medical Center, Kyoto, Japan.
Hiroshi OkunoDepartment of Urology, National Hospital Organization Kyoto Medical Center, Kyoto, Japan.
Koki MoriyoshiDepartment of Diagnostic Pathology, National Hospital Organization Kyoto Medical Center, Kyoto, Japan.
Yuto YamazakiDepartment of Pathology, Tohoku University Graduate School of Medicine, Sendai, Japan.
Hironobu SasanoDepartment of Pathology, Tohoku University Graduate School of Medicine, Sendai, Japan.
Akihiro YasodaClinical Research Institute, National Hospital Organization Kyoto Medical Center, Kyoto, Japan.
Noriko Satoh-AsaharaDepartment of Endocrinology, Metabolism, and Hypertension Research, Clinical Research Institute, National Hospital Organization Kyoto Medical Center, Kyoto, Japan.
William E RaineyDepartment of Molecular and Integrative Physiology, University of Michigan, Ann Arbor, MI, United States.
Tetsuya TagamiDepartment of Endocrinology and Metabolism, National Hospital Organization Kyoto Medical Center, Kyoto, Japan.
Kyoto Medical Center · JPTohoku University · JPUniversity of Michigan–Ann Arbor · USMichigan Center for Translational Pathology · US

Funding

MECHANISMS LEADING TO ADRENAL ZONATIONR01DK043140 · NIDDK · UNIVERSITY OF TEXAS SW MED CTR/DALLAS · PI William E Rainey · 1999 to 2026
$7.2M
Adrenal Origins of Aldosterone ExcessR01DK106618 · NIDDK · UNIVERSITY OF MICHIGAN AT ANN ARBOR · PI William E Rainey, Aaron Mark Udager · 2016 to 2026
$6.0M
NIDDK NIH HHS R01 DK043140NIDDK NIH HHS R01 DK106618
6 · The paper itself

Abstract

Double somatic mutations in

Indexed as

AdenomaAdrenocortical AdenomaHyperaldosteronismHypertensionAdultAldosteronebeta CateninCytochrome P-450 CYP11B2FemaleGTP-Binding Protein alpha SubunitsHumansMiddle AgedMutationPregnancyAldosteronebeta CateninCTNNB1 protein, humanCytochrome P-450 CYP11B2GNA11 protein, humanGTP-Binding Protein alpha Subunitsaldosterone-producing adenomaCTNNB1CYP11B2GNA11primary aldosteronismsomatic mutation

Identifiers

PMID38505749
PMCPMC10948454
OpenAlexW4392468360

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.