Article in Addiction biology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
0.5field-weighted citation impact, top 38% of its field
1 · What the graph read from it
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registry
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
3 · Its place in the literature
Who cites it
1 citing paper in PubMed, 2 citations in OpenAlex.
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what money
Authors and funding
9 authors at 3 institutions in 1 country.
Michael P DuganWaggoner Center for Alcohol and Addiction Research, Department of Neuroscience, The University of Texas at Austin, Austin, Texas, USA.ORCID 0000-0001-9796-2502
Rajani MaiyaWaggoner Center for Alcohol and Addiction Research, Department of Neuroscience, The University of Texas at Austin, Austin, Texas, USA.
Caleb FleischerWaggoner Center for Alcohol and Addiction Research, Department of Neuroscience, The University of Texas at Austin, Austin, Texas, USA.
Michal BajoDepartment of Molecular Medicine, The Scripps Research Institute, La Jolla, California, USA.
Angela E SnyderDepartment of Molecular Medicine, The Scripps Research Institute, La Jolla, California, USA.
Ashwin KoduriWaggoner Center for Alcohol and Addiction Research, Department of Neuroscience, The University of Texas at Austin, Austin, Texas, USA.
Sathvik SrinivasanWaggoner Center for Alcohol and Addiction Research, Department of Neuroscience, The University of Texas at Austin, Austin, Texas, USA.
Marisa RobertoDepartment of Molecular Medicine, The Scripps Research Institute, La Jolla, California, USA.ORCID 0000-0003-0729-3961
Robert O MessingWaggoner Center for Alcohol and Addiction Research, Department of Neuroscience, The University of Texas at Austin, Austin, Texas, USA.ORCID 0000-0002-5345-4431
The University of Texas at Austin · USScripps Research Institute · USLouisiana State University Health Sciences Center New Orleans · US
Funding
Neurpsychopharmacology-Multidisciplinary TrainingT32AA007456 · NIAAA · SCRIPPS RESEARCH INSTITUTE, THE · PI MARISA ROBERTO · 1985 to 2026
$13.4M
Electrophysiology of alcohol in extended amygdelaU01AA013498 · NIAAA · SCRIPPS RESEARCH INSTITUTE, THE · PI MARISA ROBERTO · 2001 to 2026
$12.6M
Regulation of GABAA Receptors by PKC epsilon.R37AA013588 · NIAAA · ERNEST GALLO CLINIC AND RESEARCH CENTER · PI MESSING, ROBERT O. · 2003 to 2011
$3.7M
Synaptic Mechanisms underlying sex-differences in alcohol use disorderR01AA029841 · NIAAA · SCRIPPS RESEARCH INSTITUTE, THE · PI MARISA ROBERTO · 2022 to 2026
$2.0M
Regulation of GABAA Receptors by PKC epsilon.R01AA013588 · NIAAA · UNIVERSITY OF TEXAS AT AUSTIN · PI MESSING, ROBERT O. · 2002 to 2016
$1.9M
The role of BRSK1, a PKC epsilon substrate, in behavioral and physiological responses to ethanolF31AA030218 · NIAAA · UNIVERSITY OF TEXAS AT AUSTIN · PI DUGAN, MICHAEL P · 2022 to 2023
Protein kinase C epsilon (PKCε) regulates behavioural responses to ethanol and plays a role in anxiety-like behaviour, but knowledge is limited on downstream substrates of PKCε that contribute to these behaviours. We recently identified brain-specific serine/threonine-protein kinase 1 (BRSK1) as a substrate of PKCε. Here, we test the hypothesis that BRSK1 mediates responses to ethanol and anxiety-like behaviours that are also PKCε dependent. We used in vitro kinase assays to further validate BRSK1 as a substrate of PKCε and used Brsk1
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.
Brain-specific serine/threonine-protein kinase 1 is a substrate of protein kinase C epsilon involved in sex-specific ethanol and anxiety phenotypes. · full record | OpenQuestion