Evidence map›Paper›PMID 38489405›Full record

ReviewAnnals of medicine2024

Research progress on the mechanism of common inflammatory pathways in the pathogenesis and development of lymphoma.

Aorong Shi, Fen Yun, Lin Shi, Xia Liu, Yongfeng Jia

Open access · goldAbstract readReview
In one paragraph

Review in Annals of medicine, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
1.3field-weighted citation impact, top 22% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed, 3 citations in OpenAlex.

  1. Experiences and coping strategies of time toxicity in young and middle-aged patients with lymphoma: a descriptive qualitative study.Supportive care in cancer : official journal of the Multinational Association of Supportive Care in Cancer · 2026
    Article
  2. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 1 institution in 1 country.

Aorong ShiDepartment of Pathology, Basic Medical Sciences College, Inner Mongolia Medical University, Hohhot, China.
Fen YunDepartment of Pathology, Basic Medical Sciences College, Inner Mongolia Medical University, Hohhot, China.
Lin ShiDepartment of Pathology, Basic Medical Sciences College, Inner Mongolia Medical University, Hohhot, China.
Xia LiuDepartment of Pathology, Basic Medical Sciences College, Inner Mongolia Medical University, Hohhot, China.
Yongfeng JiaDepartment of Pathology, Basic Medical Sciences College, Inner Mongolia Medical University, Hohhot, China.
Inner Mongolia Medical University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

In recent years, the incidence and mortality rates of lymphoma have gradually increased worldwide. Tumorigenesis and drug resistance are closely related to intracellular inflammatory pathways in lymphoma. Therefore, understanding the biological role of inflammatory pathways and their abnormal activation in relation to the development of lymphoma and their selective modulation may open new avenues for targeted therapy of lymphoma. The biological functions of inflammatory pathways are extensive, and they are central hubs for regulating inflammatory responses, immune responses, and the tumour immune microenvironment. However, limited studies have investigated the role of inflammatory pathways in lymphoma development. This review summarizes the relationship between abnormal activation of common inflammatory pathways and lymphoma development to identify precise and efficient targeted therapeutic options for patients with advanced, drug-resistant lymphoma.

Indexed as

LymphomaCell Transformation, NeoplasticHumansTumor Microenvironmentimmune regulationinflammatory pathwaysLymphomastumour microenvironment

Identifiers

PMID38489405
PMCPMC10946270
OpenAlexW4392856858

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.