Evidence map›Paper›PMID 38485543›Full record

ReviewPancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.]2024

Alterations in exocrine pancreatic function after acute pancreatitis.

Joseph Bejjani, Mitchell L Ramsey, Peter J Lee, Anna Evans Phillips, Vikesh K Singh, Dhiraj Yadav, Georgios I Papachristou, Phil A Hart

Open access · hybridAbstract readReview
In one paragraph

Review in Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.], 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
1.7field-weighted citation impact, top 18% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 3 citations in OpenAlex.

  1. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 3 institutions in 1 country.

Joseph BejjaniDivision of Gastroenterology, Hepatology, and Nutrition, The Ohio State University Wexner Medical Center, Columbus, OH, USA.
Mitchell L RamseyDivision of Gastroenterology, Hepatology, and Nutrition, The Ohio State University Wexner Medical Center, Columbus, OH, USA.
Peter J LeeDivision of Gastroenterology, Hepatology, and Nutrition, The Ohio State University Wexner Medical Center, Columbus, OH, USA.
Anna Evans PhillipsDivision of Gastroenterology, University of Pittsburgh Medical Center, Pittsburgh, PA, USA.
Vikesh K SinghDivision of Gastroenterology, Johns Hopkins Medical Institutions, Baltimore, MD, USA.
Dhiraj YadavDivision of Gastroenterology, University of Pittsburgh Medical Center, Pittsburgh, PA, USA.
Georgios I PapachristouDivision of Gastroenterology, Hepatology, and Nutrition, The Ohio State University Wexner Medical Center, Columbus, OH, USA.
Phil A HartDivision of Gastroenterology, Hepatology, and Nutrition, The Ohio State University Wexner Medical Center, Columbus, OH, USA. Electronic address: Philip.hart@osumc.edu.
The Ohio State University Wexner Medical Center · USUniversity of Pittsburgh Medical Center · USJohns Hopkins University · US

Funding

UPMC Clinical Center for the Study of Diabetes After Acute PancreatitisU01DK127377 · NIDDK · UNIVERSITY OF PITTSBURGH AT PITTSBURGH · PI Frederico G. S. Toledo, Dhiraj Yadav · 2020 to 2026
$2.4M
Epidemiology and Pathophysiology of Acute Pancreatitis-related Diabetes MellitusU01DK127388 · NIDDK · OHIO STATE UNIVERSITY · PI Kathleen Marie Dungan, Philip A. Hart · 2020 to 2026
$2.4M
Clinical, Radiologic and Biochemical Factors Related to Diabetes Development after Acute PancreatitisU01DK127400 · NIDDK · JOHNS HOPKINS UNIVERSITY · PI Rita R Kalyani, ZHAOLI SUN · 2020 to 2026
$2.3M
NIDDK NIH HHS U01 DK127377NIDDK NIH HHS U01 DK127388NIDDK NIH HHS U01 DK127400
6 · The paper itself

Abstract

Exocrine pancreatic dysfunction (EPD) is a malabsorptive complication of pancreatic disorders that can lead to a host of symptoms ranging from flatulence to diarrhea and contribute to weight loss and metabolic bone disease. It is increasingly recognized to occur after acute pancreatitis (AP), including episodes with mild severity. The risk of developing EPD after AP is influenced by a range of factors, including the degree of acinar cell destruction and inflammation during AP, and persistent structural derangements following AP. In this article, we discuss the epidemiology, pathophysiology, and clinical management of EPD after AP while highlighting key knowledge gaps.

Indexed as

Pancreas, ExocrinePancreatitisAcute DiseaseExocrine Pancreatic InsufficiencyHumansExocrine pancreatic dysfunctionExocrine pancreatic insufficiencyPancreatic enzymes

Identifiers

PMID38485543
PMCPMC11215795
OpenAlexW4392546528

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.