Zebrafish CCNF and FUS Mediate Stress-Specific Motor Responses.

Yagiz Alp Aksoy, Alexander J Cole, Wei Deng, Daniel Hesselson

Open access · goldAbstract read

In one paragraph

Article in Cells, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it

0cells of the map it votes in

1citing papers in PubMed

0.4field-weighted citation impact, top 43% of its field

1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 1 citations in OpenAlex.

The Wiskott-Aldrich Syndrome protein (WASp) contribution to microglial phagocytic function and neurodevelopmental support.Journal of neuroinflammation · 2026
Article

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 1 country.

Yagiz Alp AksoySydney Medical School, Faculty of Medicine and Health, The University of Sydney, Sydney, NSW 2006, Australia.

Alexander J ColeCentenary Institute, Faculty of Medicine and Health, The University of Sydney, Sydney, NSW 2006, Australia.

Wei DengSchool of Biomedical Engineering, University of Technology Sydney, Ultimo, NSW 2007, Australia.ORCID 0000-0002-9413-0978

Daniel HesselsonCentenary Institute, Faculty of Medicine and Health, The University of Sydney, Sydney, NSW 2006, Australia.ORCID 0000-0002-8675-9426

The University of Sydney · AUUniversity of Technology Sydney · AU

Funding

Ames Research Center NASA DP140103233NHMRC APP1063981NHMRC APP1095215

6 · The paper itself

Abstract

Amyotrophic lateral sclerosis (ALS) is a devastating neurodegenerative disease characterized by the degeneration of motor neurons. Mutations in the cyclin F (

Indexed as

Amyotrophic Lateral SclerosisCyclinsNeurodegenerative DiseasesRNA-Binding Protein FUSZebrafishAnimalsHumansMotor NeuronsProteinsCCNF protein, humanCyclinsFUS protein, humanProteinsRNA-Binding Protein FUSALS pathogenesisAmyotrophic Lateral Sclerosis (ALS)CCNF geneCRISPRCRISPR/Cas9 genome editingendoplasmic reticulum stressFUS genegenetic mutations in ALSin vivo genome editingmotor function analysismotor neuron developmentneurodegenerative disease researchoxidative stress responsestress-induced motor impairmentTALENTALEN-mediated genome editingZebrafish embryonic developmentZebrafish models

Identifiers

PMID38474336

DOI10.3390/cells13050372

PMCPMC10930717

OpenAlexW4392003905

What OpenQuestion holds

Textmetadata

LicenceCC BY

Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.