Evidence map›Paper›PMID 38444380›Full record

ReviewIranian biomedical journal2024

Celiac Disease: A Review from Genetic to Treatment.

Erfaneh Jafari, Niloufar Soleymani, Masoud Hamidi, Azar Rahi, Akram Rezaei, Reza Azizian

Abstract readReview
PubMed Full text at PMC Publisher
In one paragraph

Review in Iranian biomedical journal, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Erfaneh JafariPediatric Infectious Disease Research Center, Tehran University of Medical Sciences, Tehran, Iran.
Niloufar SoleymaniDepartment of Food Hygiene, Islamic Azad University (Science and Research Branch), Tehran, Iran.
Masoud HamidiÉcole Polytechnique de Bruxelles-BioMatter Unit, Université Libre de Bruxelles (ULB), Brussels, Belgium.
Azar RahiDepartment of Pathobiology, School of Public Health, Tehran University of Medical Sciences, Tehran, Iran.
Akram RezaeiDepartment of Microbiology, School of Medicine, Tehran University of Medical Sciences, Tehran, Iran.
Reza AzizianPediatric Infectious Disease Research Center, Tehran University of Medical Sciences, Tehran, Iran.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Celiac disease (CD) is a complex disorder influenced by genetic and environmental factors. When people with a genetic predisposition to CD consume gluten, an inflammatory response is triggered in the small intestine, and this reaction can be alleviated by the elimination of gluten from the diet. The clinical manifestations of CD vary greatly from person to person and begin at a young age or in adulthood. Influence of genetic factors on CD development is evident in carriers of the DQ2 and/or DQ8 allele. HLA genotypes are associated with gut colonization by bacteria, particularly in individuals suffering from CD. In addition, beneficial gut microbes are crucial for the production of DPP-4, which plays a key role in immune function, as well as metabolic and intestinal health. Therefore, probiotics have been recommended as a complementary food supplement in CD.

Indexed as

Celiac DiseaseAllelesGenetic Predisposition to DiseaseGenotypeGlutensHumansGlutensCeliac diseaseDipeptidyl peptidase 4HLA-DQ antigensProbiotics

Identifiers

PMID38444380
PMCPMC10994635

What OpenQuestion holds

Textmetadata
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.