Evidence map›Paper›PMID 38409266›Full record

ArticleNature communications2024

A distinct class of pan-cancer susceptibility genes revealed by an alternative polyadenylation transcriptome-wide association study.

Hui Chen, Zeyang Wang, Lihai Gong, Qixuan Wang, Wenyan Chen, Jia Wang, Xuelian Ma, Ruofan Ding, Xing Li, Xudong Zou and 7 more

Open access · goldAbstract read
In one paragraph

Article in Nature communications, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 12 papers.

0numbers the graph read from it
0cells of the map it votes in
12citing papers in PubMed
3.5field-weighted citation impact, top 7% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

12 citing papers in PubMed, 15 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

17 authors at 5 institutions in 4 countries.

Hui Chen *Institute of Systems and Physical Biology, Shenzhen Bay Laboratory, Shenzhen, 518055, China.
Zeyang Wang *Institute of Systems and Physical Biology, Shenzhen Bay Laboratory, Shenzhen, 518055, China.ORCID 0000-0001-5735-0675
Lihai GongInstitute of Systems and Physical Biology, Shenzhen Bay Laboratory, Shenzhen, 518055, China.ORCID 0009-0007-0880-8177
Qixuan WangInstitute of Systems and Physical Biology, Shenzhen Bay Laboratory, Shenzhen, 518055, China.ORCID 0009-0007-2736-4639
Wenyan ChenInstitute of Systems and Physical Biology, Shenzhen Bay Laboratory, Shenzhen, 518055, China.
Jia WangInstitute of Molecular Physiology, Shenzhen Bay Laboratory, Shenzhen, 518055, China.
Xuelian MaInstitute of Systems and Physical Biology, Shenzhen Bay Laboratory, Shenzhen, 518055, China.
Ruofan DingInstitute of Systems and Physical Biology, Shenzhen Bay Laboratory, Shenzhen, 518055, China.
Xing LiInstitute of Systems and Physical Biology, Shenzhen Bay Laboratory, Shenzhen, 518055, China.
Xudong ZouInstitute of Systems and Physical Biology, Shenzhen Bay Laboratory, Shenzhen, 518055, China.ORCID 0000-0002-2958-0438
Mireya PlassGene Regulation of Cell Identity Group, Regenerative Medicine Program, Bellvitge Institute for Biomedical Research (IDIBELL), L'Hospitalet de Llobregat, Barcelona, 08908, Spain.ORCID 0000-0002-9891-2723
Cheng LianDepartment of Biochemistry and Molecular Biology of School of Basic Medical Sciences, Shanghai Medical College of Fudan University, Shanghai, 200032, China.
Ting NiState Key Laboratory of Genetic Engineering, Collaborative Innovation Center of Genetics and Development, Human Phenome Institute, School of Life Sciences and Huashan Hospital, Fudan University, Shanghai, 200438, China.ORCID 0000-0001-7007-1072
Gong-Hong WeiDepartment of Biochemistry and Molecular Biology of School of Basic Medical Sciences, Shanghai Medical College of Fudan University, Shanghai, 200032, China.ORCID 0000-0001-6546-9334
Wei LiDivision of Computational Biomedicine, Department of Biological Chemistry, School of Medicine, The University of California, Irvine, CA, 92697, USA. wei.li@uci.edu.ORCID 0000-0001-9931-5990
Lin DengInstitute of Molecular Physiology, Shenzhen Bay Laboratory, Shenzhen, 518055, China. denglin@szbl.ac.cn.ORCID 0000-0003-4483-1909
Lei LiInstitute of Systems and Physical Biology, Shenzhen Bay Laboratory, Shenzhen, 518055, China. lei.li@szbl.ac.cn.ORCID 0000-0003-3924-2544
Shenzhen Bay Laboratory · CNShanghai Medical College of Fudan University · CNFudan University · CNInstitut d'Investigació Biomédica de Bellvitge · ESUniversity of California, Irvine · US

Funding

Univ.of Calif., Irvine Cancer Center Support GrantP30CA062203 · NCI · UNIVERSITY OF CALIFORNIA-IRVINE · PI Melanie Funes · 1994 to 2026
$57.9M
NCI NIH HHS P30 CA062203
6 · The paper itself

Abstract

Alternative polyadenylation plays an important role in cancer initiation and progression; however, current transcriptome-wide association studies mostly ignore alternative polyadenylation when identifying putative cancer susceptibility genes. Here, we perform a pan-cancer 3' untranslated region alternative polyadenylation transcriptome-wide association analysis by integrating 55 well-powered (n > 50,000) genome-wide association studies datasets across 22 major cancer types with alternative polyadenylation quantification from 23,955 RNA sequencing samples across 7,574 individuals. We find that genetic variants associated with alternative polyadenylation are co-localized with 28.57% of cancer loci and contribute a significant portion of cancer heritability. We further identify 642 significant cancer susceptibility genes predicted to modulate cancer risk via alternative polyadenylation, 62.46% of which have been overlooked by traditional expression- and splicing- studies. As proof of principle validation, we show that alternative alleles facilitate 3' untranslated region lengthening of CRLS1 gene leading to increased protein abundance and promoted proliferation of breast cancer cells. Together, our study highlights the significant role of alternative polyadenylation in discovering new cancer susceptibility genes and provides a strong foundational framework for enhancing our understanding of the etiology underlying human cancers.

Indexed as

NeoplasmsTranscriptome3' Untranslated RegionsGene Expression ProfilingGenome-Wide Association StudyHumansPolyadenylation3' Untranslated Regions

Identifiers

PMID38409266
PMCPMC10897204
OpenAlexW4392157338

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.