Evidence map›Paper›PMID 38386218›Full record

ReviewMolecular and cellular biochemistry2024

Role of AMP deaminase in diabetic cardiomyopathy.

Tetsuji Miura, Hidemichi Kouzu, Masaya Tanno, Yuki Tatekoshi, Atsushi Kuno

Abstract readReview
PubMed Publisher
In one paragraph

Review in Molecular and cellular biochemistry, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
0.8field-weighted citation impact, top 28% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed, 2 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 2 institutions in 1 country.

Tetsuji MiuraDepartment of Cardiovascular, Renal and Metabolic Medicine, Sapporo Medical University School of Medicine, Sapporo, Japan. miura@sapmed.ac.jp.ORCID http://orcid.org/0000-0001-5024-7085
Hidemichi KouzuDepartment of Cardiovascular, Renal and Metabolic Medicine, Sapporo Medical University School of Medicine, Sapporo, Japan.ORCID http://orcid.org/0000-0001-7815-1464
Masaya TannoDepartment of Cardiovascular, Renal and Metabolic Medicine, Sapporo Medical University School of Medicine, Sapporo, Japan.ORCID http://orcid.org/0000-0003-0845-7497
Yuki TatekoshiDepartment of Pharmacology, Sapporo Medical University School of Medicine, Sapporo, Japan.ORCID http://orcid.org/0000-0001-7345-0158
Atsushi KunoDepartment of Pharmacology, Sapporo Medical University School of Medicine, Sapporo, Japan.ORCID http://orcid.org/0000-0001-6873-1297
Sapporo Medical University · JPHokkaido University of Science · JP

Funding

Japan Society for the Promotion of Science 21K08035Japan Society for the Promotion of Science 21K08110Japan Society for the Promotion of Science 23K06152Japan Society for the Promotion of Science 23K07481
6 · The paper itself

Abstract

Diabetes mellitus is one of the major causes of ischemic and nonischemic heart failure. While hypertension and coronary artery disease are frequent comorbidities in patients with diabetes, cardiac contractile dysfunction and remodeling occur in diabetic patients even without comorbidities, which is referred to as diabetic cardiomyopathy. Investigations in recent decades have demonstrated that the production of reactive oxygen species (ROS), impaired handling of intracellular Ca

Indexed as

AMP DeaminaseDiabetic CardiomyopathiesAnimalsCalciumHumansReactive Oxygen SpeciesAMP DeaminaseCalciumReactive Oxygen SpeciesAdenine nucleotidesDiabetic cardiomyopathyEnergy metabolismMitochondria-associated membraneReactive oxygen speciesXanthine oxidoreductase

Identifiers

PMID38386218
OpenAlexW4392080692

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.