Evidence map›Paper›PMID 38368933›Full record

ArticlePhilosophical transactions of the Royal Society of London. Series B, Biological sciences2024

Functional characterization of

Elnaz Fazeli, Elham Fazeli, Petr Fojtík, Henne Holstege, Olav M Andersen

Open access · hybridAbstract read
In one paragraph

Article in Philosophical transactions of the Royal Society of London. Series B, Biological sciences, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
3.0field-weighted citation impact, top 9% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 14 citations in OpenAlex.

  1. The Alzheimer's disease risk gene SORL1 is a regulator of excitatory neuronal function.Alzheimer's & dementia : the journal of the Alzheimer's Association · 2026
    Article
  2. Article
  3. Article
  4. Article
  5. Article
  6. Familial Alzheimer's disease mutation identifies novel role of SORLA in release of neurotrophic exosomes.Alzheimer's & dementia : the journal of the Alzheimer's Association · 2025
    Article
  7. Article
  8. Transethnic analysis identifies SORL1 variants and haplotypes protective against Alzheimer's disease.Alzheimer's & dementia : the journal of the Alzheimer's Association · 2025
    Article
  9. TheProceedings of the National Academy of Sciences of the United States of America · 2024
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 2 institutions in 2 countries.

Elnaz FazeliDepartment of Biomedicine, Danish Research Institute of Translational Neuroscience, Aarhus University, Aarhus 8000, Denmark.
Elham FazeliDepartment of Biomedicine, Danish Research Institute of Translational Neuroscience, Aarhus University, Aarhus 8000, Denmark.
Petr FojtíkDepartment of Biomedicine, Danish Research Institute of Translational Neuroscience, Aarhus University, Aarhus 8000, Denmark.ORCID 0000-0003-0188-0022
Henne HolstegeDepartment of Human Genetics, Amsterdam University Medical Center, Amsterdam Neurosocience, Vrije Universiteit, 1081 HV Amsterdam, The Netherlands.
Olav M AndersenDepartment of Biomedicine, Danish Research Institute of Translational Neuroscience, Aarhus University, Aarhus 8000, Denmark.ORCID 0000-0003-4226-3354
Aarhus University · DKVrije Universiteit Amsterdam · NL

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

SORLA, the protein encoded by the

Indexed as

Alzheimer DiseaseGenetic Predisposition to DiseaseHumansLDL-Receptor Related ProteinsMembrane Transport ProteinsMutationVirulenceLDL-Receptor Related ProteinsMembrane Transport ProteinsSORL1 protein, humanAlzheimer's diseasepathogenic variantsreceptor maturationSORL1SORLA

Identifiers

PMID38368933
PMCPMC10874698
OpenAlexW4391918767

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.