Evidence map›Paper›PMID 38357442›Full record

ArticleFrontiers in cellular and infection microbiology2024

Upregulation of ATP6V0D2 benefits intracellular survival of

Jing Hong, Budhaditya Mukherjee, Chizu Sanjoba, Junya Yamagishi, Yasuyuki Goto

Open access · goldAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Frontiers in cellular and infection microbiology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
0.5field-weighted citation impact, top 35% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed, 1 citations in OpenAlex.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 3 institutions in 2 countries.

Jing HongLaboratory of Molecular Immunology, Graduate School of Agricultural and Life Sciences, The University of Tokyo, Tokyo, Japan.
Budhaditya MukherjeeSchool of Medical Science and Technology, Indian Institute of Technology Kharagpur, Kharagpur, India.
Chizu SanjobaLaboratory of Molecular Immunology, Graduate School of Agricultural and Life Sciences, The University of Tokyo, Tokyo, Japan.
Junya YamagishiInternational Collaboration Unit, International Institute for Zoonosis Control, Hokkaido University, Sapporo, Japan.
Yasuyuki GotoLaboratory of Molecular Immunology, Graduate School of Agricultural and Life Sciences, The University of Tokyo, Tokyo, Japan.
The University of Tokyo · JPHokkaido University · JPIndian Institute of Technology Kharagpur · IN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Visceral leishmaniasis (VL) is the most severe type of leishmaniasis which is caused by infection of

Indexed as

Leishmania donovaniLeishmaniasis, VisceralAnimalsErythrocytesIronMacrophagesMiceMice, Inbred BALB CUp-RegulationVacuolar Proton-Translocating ATPasesAtp6v0d2 protein, mouseIronVacuolar Proton-Translocating ATPasesATP6V0D2hemophagocytosisironLeishmaniamacrophagemultinucleated giant cell (MGC)

Identifiers

PMID38357442
PMCPMC10864549
OpenAlexW4391400050

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.