Evidence map›Paper›PMID 38348775›Full record

ArticleJournal of the American Heart Association2024

Effect of Accessory Renal Arteries on Essential Hypertension and Related Mechanisms.

Fengyuan Wu, Xiaoyang Yuan, Kaiwen Sun, Ying Zhang, Lianxin Zhu, Cuiping Bai, Yunpeng Cheng, Yan Lu, Yinong Jiang, Wei Song

Abstract read
In one paragraph

Article in Journal of the American Heart Association, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed.

  1. Trial
  2. Article
  3. Overcoming variable response in renal denervation: Key strategies for reliable clinical implementation.Hypertension research : official journal of the Japanese Society of Hypertension · 2026
    Review
  4. Article
  5. Value of multi-modal ultrasound in evaluation of the accessory renal artery.Quantitative imaging in medicine and surgery · 2025
    Article
  6. Article
  7. Review
  8. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors.

Fengyuan WuDepartment of Cardiology First Affiliated Hospital of Dalian Medical University Dalian Liaoning China.
Xiaoyang YuanDepartment of Clinical Laboratory First Affiliated Hospital of Dalian Medical University Dalian Liaoning China.
Kaiwen SunDepartment of Cardiology First Affiliated Hospital of Dalian Medical University Dalian Liaoning China.
Ying ZhangDepartment of Cardiology First Affiliated Hospital of Dalian Medical University Dalian Liaoning China.
Lianxin ZhuDepartment of Cardiology First Affiliated Hospital of Dalian Medical University Dalian Liaoning China.
Cuiping BaiDepartment of Cardiology First Affiliated Hospital of Dalian Medical University Dalian Liaoning China.
Yunpeng ChengDepartment of Cardiology First Affiliated Hospital of Dalian Medical University Dalian Liaoning China.ORCID 0000-0002-1589-1921
Yan LuDepartment of Cardiology First Affiliated Hospital of Dalian Medical University Dalian Liaoning China.
Yinong JiangDepartment of Cardiology First Affiliated Hospital of Dalian Medical University Dalian Liaoning China.
Wei SongDepartment of Cardiology First Affiliated Hospital of Dalian Medical University Dalian Liaoning China.ORCID 0000-0002-4973-7236

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundThis case-control study aimed to determine whether there were differences between patients with essential hypertension with accessory renal arteries (ARAs) and those without ARAs. METHODS AND

resultsThe enrolled patients with essential hypertension were divided into the ARA group (n=200) and control group without ARAs (n=238). After propensity matching, 394 patients (197 in each of the 2 groups), were included. The 24-hour BP (4.33/2.43 mm Hg) and daytime BP (4.48/2.61 mm Hg) of patients in the ARA group were significantly higher than those of the control group (

conclusionsARAs could contribute a higher BP of patients with essential hypertension and might promote the development of essential hypertension. The mechanism might be related to overactivation of the renin-angiotensin-aldosterone system and sympathetic nervous system.

Indexed as

HypertensionAldosteroneBlood PressureBlood Pressure Monitoring, AmbulatoryCase-Control StudiesEssential HypertensionHumansRenal ArteryAldosteroneaccessory renal arteryaldosteroneessential hypertensionreninsympathetic nervous activity

Identifiers

PMID38348775
PMCPMC11010091

What OpenQuestion holds

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LicenceCC BY-NC-ND
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.