Evidence map›Paper›PMID 38344969›Full record

ArticleJournal of clinical research in pediatric endocrinology2026

Hyperinsulinemia in Sotos Syndrome with a

Elena Lundberg, Magnus Burstedt, Irina Golovleva

Open access · goldAbstract readCase Reports
In one paragraph

Article in Journal of clinical research in pediatric endocrinology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact, top 97% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed, 0 citations in OpenAlex.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Elena LundbergUmeå University, Institute of Clinical Science, Department of Pediatrics, Umeå, SwedenORCID 0000-0001-6010-351X
Magnus BurstedtUmeå University, Institute of Medical Biosciences, Department of Medical and Clinical Genetics, Umeå, SwedenORCID 0000-0001-5550-0633
Irina GolovlevaUmeå University, Institute of Medical Biosciences, Department of Medical and Clinical Genetics, Umeå, SwedenORCID 0000-0001-8741-0616
Umeå University · SE

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Sotos syndrome belongs to the group of diseases characterised by features such as facial dysmorphism, intellectual disability, hypotonia and overgrowth. Usually, Sotos syndrome is caused by heterozygous mutations in the

Indexed as

Gene DeletionHistone-Lysine N-MethyltransferaseHyperinsulinismIntracellular Signaling Peptides and ProteinsSotos SyndromeFemaleHistone MethyltransferasesHumansInfant, NewbornMalePhenotypeHistone-Lysine N-MethyltransferaseHistone MethyltransferasesIntracellular Signaling Peptides and ProteinsNSD1 protein, humanHyperinsulinemiahypoglycaemiaNSD1overgrowthSotos syndrome

Identifiers

PMID38344969
PMCPMC12989897
OpenAlexW4391746597

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.