Evidence map›Paper›PMID 38333410›Full record

ArticleJournal of arrhythmia2024

Comparison of the electrophysiological properties of the pulmonary veins between paroxysmal and persistent atrial fibrillation.

Hitoshi Mori, Akira Hamabe, Daisuke Kawano, Tsukasa Naganuma, Mai Tahara, Yodo Gatate, Toyokazu Kimura, Hirotsugu Tabata, Ritsushi Kato

Open access · goldAbstract read
In one paragraph

Article in Journal of arrhythmia, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
1.3field-weighted citation impact, top 16% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed, 6 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 2 institutions in 1 country.

Hitoshi MoriDepartment of Cardiology Japan Self Defense Forces Central Hospital Tokyo Japan.ORCID https://orcid.org/0000-0002-1172-2995
Akira HamabeDepartment of Cardiology Japan Self Defense Forces Central Hospital Tokyo Japan.
Daisuke KawanoDepartment of Cardiology Japan Self Defense Forces Central Hospital Tokyo Japan.ORCID https://orcid.org/0000-0002-3347-6772
Tsukasa NaganumaDepartment of Cardiology Japan Self Defense Forces Central Hospital Tokyo Japan.
Mai TaharaDepartment of Cardiology Japan Self Defense Forces Central Hospital Tokyo Japan.
Yodo GatateDepartment of Cardiology Japan Self Defense Forces Central Hospital Tokyo Japan.
Toyokazu KimuraDepartment of Cardiology Japan Self Defense Forces Central Hospital Tokyo Japan.
Hirotsugu TabataDepartment of Cardiology Japan Self Defense Forces Central Hospital Tokyo Japan.
Ritsushi KatoDepartment of Cardiology Saitama Medical University, International Medical Center Hidaka Japan.ORCID https://orcid.org/0000-0002-9989-8708
Self-Defense Forces Central Hospital · JPSaitama Medical University · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Background: The role of the pulmonary veins (PVs) as triggers in atrial fibrillation (AF) is well-known; however, their detailed electrophysiological properties have not been thoroughly examined. Objective: This study aimed to investigate the electrophysiological properties of the PVs between paroxysmal AF (pAF) and persistent AF (perAF). Methods: Prior to catheter ablation in patients with pAF ( Results: Compared with perAF, the myocardial sleeves of PVs for pAF were significantly larger for all PVs. The ERP for perAF was significantly shorter than that for pAF for all PVs. The pacing threshold for perAF was significantly higher than that for pAF for the right and left superior PVs. Conclusion: In patients with perAF, a decrease in the normal myocardial sleeves and a shortening of the ERP were observed for all PVs. Those changes in the electrophysiological properties of the PVs might be related to the persistence of AF.

Indexed as

atrial fibrillationelectrophysiological studylow‐voltage areaspulmonary veinsubstrate

Identifiers

PMID38333410
PMCPMC10848593
OpenAlexW4390410995

What OpenQuestion holds

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LicenceCC BY
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.