Evidence map›Paper›PMID 38315272›Full record

ReviewBreast cancer (Tokyo, Japan)2024

Advances in the study of autophagy in breast cancer.

Tang Yu, Liu Rui, Zhao Jiumei, Li Ziwei, Hu Ying

Open access · hybridAbstract readReview
In one paragraph

Review in Breast cancer (Tokyo, Japan), 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 12 papers.

0numbers the graph read from it
0cells of the map it votes in
12citing papers in PubMed
4.3field-weighted citation impact, top 5% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

12 citing papers in PubMed, 11 citations in OpenAlex.

  1. Review
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  11. The role of autophagy in pancreatic diseases.Frontiers in pharmacology · 2024
    Review
  12. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 4 institutions in 1 country.

Tang YuThe Third Affiliated Hospital of Kunming Medical University, Kunming, China.
Liu RuiThe Third Affiliated Hospital of Kunming Medical University, Kunming, China.
Zhao JiumeiChongqing Nanchuan District People's Hospital, Chongqing, China.
Li ZiweiChongqing Health Center for Women and Children, Women and Children's Hospital of Chongqing Medical University, Chongqing, China.
Hu YingThe Second Affiliatied Hospital of Kunming Medical University and Department of Clinical Larboratory, Kunming, China. hy2002@126.com.
Kunming Medical University · CNChildren's Hospital of Chongqing Medical University · CNChongqing Dazu District People's Hospital · CNChongqing Medical University · CN

Funding

Applied Basic Research Key Project of Yunnan 2023Y0787Center for Depression Research and Clinical Care, University of Texas Southwestern Medical Center 202102AA100067Yunnan health training project of high level talents D-2018041
6 · The paper itself

Abstract

Breast cancer is the most prevalent malignant tumor among women, with a high incidence and mortality rate all year round, which seriously affects women's health. Autophagy, a well-conserved cellular process inherent in eukaryotic organisms, plays a pivotal role in degrading damaged proteins and organelles, recycling their breakdown products to aid cells in navigating stress and gradually restoring homeostatic equilibrium. Recent studies have unveiled the intricate connection between autophagy and breast cancer. Autophagy is a double-edged sword in breast cancer, demonstrating a dual role: restraining its onset and progression on one hand, while promoting its metastasis and advancement on the other. It is also because of this interrelationship between the two that regulation of autophagy in the treatment of breast cancer is now an important strategy in clinical treatment. In this article, we systematically survey the recent research findings, elucidating the multifaceted role of autophagy in breast cancer and its underlying mechanisms, with the aim of contributing new references to the clinical management of breast cancer.

Indexed as

Breast NeoplasmsAutophagyFemaleHumansProteinsProteinsAutophagyAutophagy inhibitorBreast cancerMolecular mechanismTherapy

Identifiers

PMID38315272
PMCPMC10901946
OpenAlexW4391543693

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.