Evidence map›Paper›PMID 38304423›Full record

ReviewFrontiers in cell and developmental biology2024

hiPSC-derived cardiomyocytes as a model to study the role of small-conductance Ca

Hosna Babini, Verónica Jiménez-Sábado, Ekaterina Stogova, Alia Arslanova, Mariam Butt, Saif Dababneh, Parisa Asghari, Edwin D W Moore, Thomas W Claydon, Nipavan Chiamvimonvat and 2 more

Open access · goldAbstract readReview
In one paragraph

Review in Frontiers in cell and developmental biology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
3.2field-weighted citation impact, top 8% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed, 8 citations in OpenAlex.

  1. Review
  2. Article
  3. Single-Nucleotide Polymorphisms,International journal of molecular sciences · 2025
    Review
  4. Article
  5. Review
  6. Review
  7. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors at 4 institutions in 3 countries.

Hosna Babini *Cellular and Regenerative Medicine Centre, BC Children's Hospital Research Institute, Vancouver, BC, Canada.
Verónica Jiménez-Sábado *Cellular and Regenerative Medicine Centre, BC Children's Hospital Research Institute, Vancouver, BC, Canada.
Ekaterina StogovaCellular and Regenerative Medicine Centre, BC Children's Hospital Research Institute, Vancouver, BC, Canada.
Alia ArslanovaCellular and Regenerative Medicine Centre, BC Children's Hospital Research Institute, Vancouver, BC, Canada.
Mariam ButtCellular and Regenerative Medicine Centre, BC Children's Hospital Research Institute, Vancouver, BC, Canada.
Saif DababnehCellular and Regenerative Medicine Centre, BC Children's Hospital Research Institute, Vancouver, BC, Canada.
Parisa AsghariDepartment of Cellular and Physiological Sciences, Faculty of Medicine, University of British Columbia, Vancouver, BC, Canada.
Edwin D W MooreDepartment of Cellular and Physiological Sciences, Faculty of Medicine, University of British Columbia, Vancouver, BC, Canada.
Thomas W ClaydonBiomedical Physiology and Kinesiology, Simon Fraser University, Burnaby, BC, Canada.
Nipavan ChiamvimonvatUniversity of California, Davis, Davis, CA, United States.
Leif Hove-MadsenIIB SANT PAU, and CIBERCV, Hospital de la Santa Creu i Sant Pau, Barcelona, Spain.
Glen F TibbitsCellular and Regenerative Medicine Centre, BC Children's Hospital Research Institute, Vancouver, BC, Canada.
Simon Fraser University · CAUniversity of British Columbia · CAHospital de Sant Pau · ESUniversity of California, Davis · US

Funding

Functional Interactions of Cardiac Ion ChannelsR01HL085844 · NHLBI · UNIVERSITY OF CALIFORNIA AT DAVIS · PI CHIAMVIMONVAT, NIPAVAN · 2007 to 2025
$7.3M
Translational Study of Cardiac DysfunctionR01HL085727 · NHLBI · UNIVERSITY OF CALIFORNIA AT DAVIS · PI CHIAMVIMONVAT, NIPAVAN, SIRISH, PADMINI · 2008 to 2022
$5.7M
Decoding the enigma of cardiac amplificationR01HL137228 · NHLBI · UNIVERSITY OF CALIFORNIA AT DAVIS · PI CHIAMVIMONVAT, NIPAVAN · 2017 to 2020
$2.2M
NHLBI NIH HHS R01 HL085727NHLBI NIH HHS R01 HL085844NHLBI NIH HHS R01 HL137228
6 · The paper itself

Abstract

Atrial fibrillation (AF), the most common arrhythmia, has been associated with different electrophysiological, molecular, and structural alterations in atrial cardiomyocytes. Therefore, more studies are required to elucidate the genetic and molecular basis of AF. Various genome-wide association studies (GWAS) have strongly associated different single nucleotide polymorphisms (SNPs) with AF. One of these GWAS identified the rs13376333 risk SNP as the most significant one from the 1q21 chromosomal region. The rs13376333 risk SNP is intronic to the

Indexed as

atrial fibrillationcalciumcardiac action potentialcardiomyocyteshuman induced pluripotent stem cellssingle nucleotide polymorphismssmall-conductance Ca2+-activated K+ channels

Identifiers

PMID38304423
PMCPMC10830749
OpenAlexW4390982281

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.