Evidence map›Paper›PMID 38269628›Full record

ArticleClinical cardiology2024

Contractility, ventriculoarterial coupling, and stroke work after acute myocardial infarction using CMR-derived pressure-volume loop data.

David Nordlund, Theodor Lav, Robert Jablonowski, Ardavan Khoshnood, Ulf Ekelund, Dan Atar, David Erlinge, Henrik Engblom, Håkan Arheden

Abstract read
In one paragraph

Article in Clinical cardiology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors.

David NordlundDepartment of Clinical Sciences Lund, Clinical Physiology, Lund University and Skane University Hospital, Lund, Sweden.ORCID http://orcid.org/0000-0002-6567-8459
Theodor LavDepartment of Clinical Sciences Lund, Clinical Physiology, Lund University and Skane University Hospital, Lund, Sweden.
Robert JablonowskiDepartment of Clinical Sciences Lund, Clinical Physiology, Lund University and Skane University Hospital, Lund, Sweden.
Ardavan KhoshnoodDepartment of Clinical Sciences Malmö, Emergency Medicine, Lund University, Skane University Hospital, Malmö, Sweden.
Ulf EkelundDepartment of Clinical Sciences Lund, Emergency Medicine, Lund University and Skane University Hospital, Lund, Sweden.
Dan AtarDept. of Cardiology, Oslo University Hospital Ulleval, Oslo, Norway.
David ErlingeCardiology, Department of Clinical Sciences Lund, Lund University and Skane University Hospital, Lund, Sweden.
Henrik EngblomDepartment of Clinical Sciences Lund, Clinical Physiology, Lund University and Skane University Hospital, Lund, Sweden.
Håkan ArhedenDepartment of Clinical Sciences Lund, Clinical Physiology, Lund University and Skane University Hospital, Lund, Sweden.

Funding

Hjärt-LungfondenLund University Medical Faculty FoundationRegion of Scania
6 · The paper itself

Abstract

backgroundNoninvasive left ventricular (LV) pressure-volume (PV) loops derived by cardiac magnetic resonance (CMR) have recently been shown to enable characterization of cardiac hemodynamics. Thus, such PV loops could potentially provide additional diagnostic information such as contractility, arterial elastance (E

methodsA total of 100 patients with acute MI and 75 controls were included in the study. All patients underwent CMR 2-6 days after MI including assessment of myocardium at risk (MaR) and infarct size (IS). Noninvasive PV loops were generated from CMR derived LV volumes and brachial blood pressure measurements. The following variables were quantified: Maximal elastance (E

resultsAll PV-loop variables were significantly different in MI patients compared to healthy volunteers, including contractility (E

conclusionsNoninvasive PV-loops provide physiological information beyond conventional diagnostic variables, such as ejection fraction, early after MI, including measures of contractility, ventriculoarterial coupling, and SW.

Indexed as

Myocardial InfarctionStrokeHeartHumansMagnetic Resonance ImagingMagnetic Resonance Spectroscopycontractilitycouplingefficiencyelastancemyocardium at riskstroke work

Identifiers

PMID38269628
PMCPMC10790509

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.