Evidence map›Paper›PMID 38254653›Full record

ArticleBiomolecules2023

Increased Risk for Atrial Alternans in Rabbit Heart Failure: The Role of Ca

Giedrius Kanaporis, Lothar A Blatter

Open access · goldAbstract read
In one paragraph

Article in Biomolecules, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
0.2field-weighted citation impact, top 38% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 1 citations in OpenAlex.

  1. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 1 institution in 1 country.

Giedrius KanaporisDepartment of Physiology & Biophysics, Rush University Medical Center, Chicago, IL 60612, USA.ORCID 0000-0003-3593-1182
Lothar A BlatterDepartment of Physiology & Biophysics, Rush University Medical Center, Chicago, IL 60612, USA.ORCID 0000-0003-3409-8678
Rush University Medical Center · US

Funding

Limiting Pathological Calcium Induced Calcium Release in HeartR01HL057832 · NHLBI · RUSH UNIVERSITY MEDICAL CENTER · PI FILL, MICHAEL, GILLESPIE, DIRK · 1997 to 2024
$9.0M
Atrial Excitation-Contraction Coupling, Calcium Signaling and Electro-Mechanical AlternansR01HL164453 · NHLBI · RUSH UNIVERSITY MEDICAL CENTER · PI BANACH, KATHRIN, KANAPORIS, GIEDRIUS · 2022 to 2025
$2.8M
IP3 receptor, NOX2 and calcium signaling domains in atrial physiology and pathophysiologyR01HL155762 · NHLBI · RUSH UNIVERSITY MEDICAL CENTER · PI BANACH, KATHRIN · 2022 to 2025
$2.7M
Pathophysiological Regulation of Atrial Alternans and Atrial FibrillationR01HL132871 · NHLBI · RUSH UNIVERSITY MEDICAL CENTER · PI BANACH, KATHRIN, BLATTER, LOTHAR A · 2017 to 2020
$1.6M
Pathophysiological Regulation of Atrial Myocyte Excitation-Contraction Coupling and Calcium SignalingR01HL134781 · NHLBI · RUSH UNIVERSITY MEDICAL CENTER · PI BLATTER, LOTHAR A · 2017 to 2020
$1.5M
NHLBI NIH HHS R01 HL057832NHLBI NIH HHS R01 HL132871NHLBI NIH HHS R01 HL134781NHLBI NIH HHS R01 HL155762NHLBI NIH HHS R01 HL164453
6 · The paper itself

Abstract

Heart failure (HF) increases the probability of cardiac arrhythmias, including atrial fibrillation (AF), but the mechanisms linking HF to AF are poorly understood. We investigated disturbances in Ca

Indexed as

Atrial FibrillationCalcium-Calmodulin-Dependent Protein Kinase Type 2Heart FailureInositol 1,4,5-TrisphosphatePeptide HormonesAngiotensin IIAnimalsCalmodulinHeart AtriaInositol 1,4,5-Trisphosphate ReceptorsRabbitsAngiotensin IICalcium-Calmodulin-Dependent Protein Kinase Type 2CalmodulinInositol 1,4,5-TrisphosphateInositol 1,4,5-Trisphosphate ReceptorsPeptide HormonesalternansarrhythmiaatriaCa2+/calmodulin-dependent kinase IIcalciumheart failureinositol-trisphosphate

Identifiers

PMID38254653
PMCPMC10813785
OpenAlexW4390459765

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.