Evidence map›Paper›PMID 38216787›Full record

ReviewEMBO reports2024

Immunosurveillance encounters cancer metabolism.

Yu-Ming Chuang, Sheue-Fen Tzeng, Ping-Chih Ho, Chin-Hsien Tsai

Open access · diamondAbstract readReview
In one paragraph

Review in EMBO reports, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 21 papers.

0numbers the graph read from it
0cells of the map it votes in
21citing papers in PubMed
7.7field-weighted citation impact, top 2% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

21 citing papers in PubMed, 27 citations in OpenAlex.

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  18. Cold and hot tumors: from molecular mechanisms to targeted therapy.Signal transduction and targeted therapy · 2024
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 2 countries.

Yu-Ming Chuang *Department of Fundamental Oncology, University of Lausanne, Lausanne, Switzerland.
Sheue-Fen Tzeng *Graduate Institute of Life Sciences, National Defense Medical Center, Taipei, Taiwan.ORCID http://orcid.org/0000-0003-1126-6763
Ping-Chih HoDepartment of Fundamental Oncology, University of Lausanne, Lausanne, Switzerland. ping-chih.ho@unil.ch.ORCID http://orcid.org/0000-0003-3078-3774
Chin-Hsien TsaiGraduate Institute of Life Sciences, National Defense Medical Center, Taipei, Taiwan. tch@mail.ndmctsgh.edu.tw.ORCID http://orcid.org/0000-0001-5278-2321
Ludwig Cancer Research · CHNational Defense Medical Center · TW

Funding

EC | ERC | HORIZON EUROPE European Research Council (ERC) 802773MOST | Institute for Information Industry, Ministry of Science and Technology, Taiwan (III) 111-2314-B-016-004National Defense Medical Center (NDMC) MND-MAB-D-112095Schweizerischer Nationalfonds zur Förderung der Wissenschaftlichen Forschung (SNF) 31003A_182470Schweizerischer Nationalfonds zur Förderung der Wissenschaftlichen Forschung (SNF) IZLCZO_206083
6 · The paper itself

Abstract

Tumor cells reprogram nutrient acquisition and metabolic pathways to meet their energetic, biosynthetic, and redox demands. Similarly, metabolic processes in immune cells support host immunity against cancer and determine differentiation and fate of leukocytes. Thus, metabolic deregulation and imbalance in immune cells within the tumor microenvironment have been reported to drive immune evasion and to compromise therapeutic outcomes. Interestingly, emerging evidence indicates that anti-tumor immunity could modulate tumor heterogeneity, aggressiveness, and metabolic reprogramming, suggesting that immunosurveillance can instruct cancer progression in multiple dimensions. This review summarizes our current understanding of how metabolic crosstalk within tumors affects immunogenicity of tumor cells and promotes cancer progression. Furthermore, we explain how defects in the metabolic cascade can contribute to developing dysfunctional immune responses against cancers and discuss the contribution of immunosurveillance to these defects as a feedback mechanism. Finally, we highlight ongoing clinical trials and new therapeutic strategies targeting cellular metabolism in cancer.

Indexed as

NeoplasmsEnergy MetabolismHumansMetabolic Networks and PathwaysMonitoring, ImmunologicTumor MicroenvironmentCancer EvolutionImmunoeditingImmunometabolism

Identifiers

PMID38216787
PMCPMC10897436
OpenAlexW4390796731

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.