Evidence map›Paper›PMID 38193901›Full record

ArticleeLife2024

Notch signaling and Bsh homeodomain activity are integrated to diversify

Chundi Xu, Tyler B Ramos, Owen J Marshall, Chris Q Doe

Open access · goldAbstract read
In one paragraph

Article in eLife, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
1.8field-weighted citation impact, top 15% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed, 12 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Article
  5. Vertebrate and Invertebrate Animal Models for the Study of Down Syndrome.International journal of molecular sciences · 2025
    Review
  6. Review
  7. Review
  8. A Closer Look at Histamine inInternational journal of molecular sciences · 2024
    Review
  9. Article
  10. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 2 countries.

Chundi XuInstitute of Neuroscience, Howard Hughes Medical Institute, University of Oregon, Eugene, United States.ORCID https://orcid.org/0000-0002-1056-8893
Tyler B RamosInstitute of Neuroscience, Howard Hughes Medical Institute, University of Oregon, Eugene, United States.
Owen J MarshallMenzies Institute for Medical Research, University of Tasmania, Hobart, Australia.
Chris Q DoeInstitute of Neuroscience, Howard Hughes Medical Institute, University of Oregon, Eugene, United States.ORCID https://orcid.org/0000-0001-5980-8029
Howard Hughes Medical Institute · USUniversity of Tasmania · AU

Funding

GENETIC AND MOLECULAR STUDIES OF NEUROGENESISR01HD027056 · NICHD · UNIVERSITY OF OREGON · PI Chris Q Doe · 1989 to 2026
$5.0M
NICHD NIH HHS R01 HD027056
6 · The paper itself

Abstract

Notch signaling is an evolutionarily conserved pathway for specifying binary neuronal fates, yet how it specifies different fates in different contexts remains elusive. In our accompanying paper, using the

Indexed as

DrosophilaDrosophila ProteinsReceptors, NotchTranscription FactorsAnimalsChromatinGenomicsNeuronsPOU Domain FactorsChromatinDrosophila ProteinsPdm3 protein, DrosophilaPOU Domain FactorsReceptors, NotchTranscription FactorsBshchromatin landscapedevelopmental biologyD. melanogasterhomeodomainlaminaneuronal fatenotch

Identifiers

PMID38193901
PMCPMC10945509
OpenAlexW4387253638

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.