Evidence map›Paper›PMID 38102518›Full record

ReviewMolecular neurobiology2024

Non-Coding RNAs and Neurodegenerative Diseases: Information of their Roles in Apoptosis.

Mehrdad Valizadeh, Ehsan Derafsh, Fahimeh Abdi Abyaneh, Sayedeh Kiana Parsamatin, Fatemeh Zahra Rashidi Noshabad, Azam Alinaghipour, Zahra Yaghoobi, Abdolkarim Talebi Taheri, Ehsan Dadgostar, Michael Aschner and 3 more

Abstract readReview
PubMed Publisher
In one paragraph

Review in Molecular neurobiology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
1.2field-weighted citation impact, top 18% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 5 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors at 7 institutions in 3 countries.

Mehrdad ValizadehShahid Beheshti University of Medical Sciences, Tehran, Iran.
Ehsan DerafshWindsor University School of Medicine, Cayon, Canada.
Fahimeh Abdi AbyanehSchool of Medicine, Kashan University of Medical Sciences, Kashan, Iran.
Sayedeh Kiana ParsamatinDepartment of Neurology, School of Medicine, Isfahan University of Medical Sciences, Isfahan, Iran.
Fatemeh Zahra Rashidi NoshabadStudent Research Committee, Kashan University of Medical Sciences, Kashan, IR, Iran.
Azam AlinaghipourSchool of Medical Sciences, Yazd Branch, Islamic Azad University, Yazd, Iran.
Zahra YaghoobiElectrophysiology Research Center, Neuroscience Institute, Tehran University of Medical Sciences, Tehran, IR, Iran.
Abdolkarim Talebi TaheriStudent Research Committee, School of Medicine, Shahid Beheshti University of Medical Sciences, Tehran, Iran.
Ehsan DadgostarBehavioral Sciences Research Center, Isfahan University of Medical Sciences, Isfahan, IR, Iran.
Michael AschnerDepartment of Molecular Pharmacology, Albert Einstein College of Medicine, Bronx, NY, 10461, USA.
Hamed MirzaeiResearch Center for Biochemistry and Nutrition in Metabolic Diseases, Kashan University of Medical Sciences, Kashan, IR, Iran. h.mirzaei2002@gmail.com.
Omid Reza TamtajiElectrophysiology Research Center, Neuroscience Institute, Tehran University of Medical Sciences, Tehran, IR, Iran. tamtaji.or@gmail.com.ORCID http://orcid.org/0000-0003-2492-3996
Fatemeh NabavizadehElectrophysiology Research Center, Neuroscience Institute, Tehran University of Medical Sciences, Tehran, IR, Iran.
Kashan University of Medical Sciences · IRTehran University of Medical Sciences · IRIsfahan University of Medical Sciences · IRShahid Beheshti University of Medical Sciences · IRAlbert Einstein College of Medicine · USIslamic Azad University, Yazd · IRUniversity of Windsor · CA

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Apoptosis can be known as a key factor in the pathogenesis of neurodegenerative disorders. In disease conditions, the rate of apoptosis expands and tissue damage may become apparent. Recently, the scientific studies of the non-coding RNAs (ncRNAs) has provided new information of the molecular mechanisms that contribute to neurodegenerative disorders. Numerous reports have documented that ncRNAs have important contributions to several biological processes associated with the increase of neurodegenerative disorders. In addition, microRNAs (miRNAs), circular RNAs (circRNAs), as well as, long ncRNAs (lncRNAs) represent ncRNAs subtypes with the usual dysregulation in neurodegenerative disorders. Dysregulating ncRNAs has been associated with inhibiting or stimulating apoptosis in neurodegenerative disorders. Therefore, this review highlighted several ncRNAs linked to apoptosis in neurodegenerative disorders. CircRNAs, lncRNAs, and miRNAs were also illustrated completely regarding the respective signaling pathways of apoptosis.

Indexed as

ApoptosisNeurodegenerative DiseasesRNA, UntranslatedAnimalsHumansMicroRNAsRNA, CircularSignal TransductionMicroRNAsRNA, CircularRNA, UntranslatedApoptosisCircular RNALong non-coding RNAMicroRNANeurodegenerative disorders

Identifiers

PMID38102518
OpenAlexW4389805210

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.