Evidence map›Paper›PMID 38045957›Full record

ReviewFrontiers in public health2023

Cigarette smoking and air pollution exposure and their effects on cardiovascular diseases.

Manthar Ali Mallah, Tahmina Soomro, Mukhtiar Ali, Sobia Noreen, Nafeesa Khatoon, Akriti Kafle, Feifei Feng, Wei Wang, Muhammad Naveed, Qiao Zhang

Open access · goldAbstract readReview
In one paragraph

Review in Frontiers in public health, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 33 papers.

0numbers the graph read from it
0cells of the map it votes in
33citing papers in PubMed
8.0field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

33 citing papers in PubMed, 51 citations in OpenAlex.

  1. Review
  2. Review
  3. Article
  4. Article
  5. Cardioprotective Role of CalhexCell biochemistry and biophysics · 2026
    Review
  6. Article
  7. Article
  8. Article
  9. Review
  10. Article
  11. Article
  12. Article
  13. Impact of fasting on lipid profile assessment of Brazilian adults.Archives of endocrinology and metabolism · 2025
    Article
  14. Article
  15. Article
  16. Article
  17. Article
  18. Review
  19. Observational
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 5 institutions in 4 countries.

Manthar Ali MallahCollege of Public Health, Zhengzhou University, Zhengzhou, China.
Tahmina SoomroDepartment of Sociology, Shah Abdul Latif University, Khairpur, Pakistan.
Mukhtiar AliDepartment of Chemical Engineering, Quaid-e-Awam University of Engineering, Science and Technology, Nawabshah, Sindh, Pakistan.
Sobia NoreenDepartment of Pharmaceutics Technology, Institute of Pharmacy, University of Innsbruck, Insbruck, Austria.
Nafeesa KhatoonCollege of Public Health, Zhengzhou University, Zhengzhou, China.
Akriti KafleSchool of Nursing, Zhengzhou University, Zhengzhou, China.
Feifei FengCollege of Public Health, Zhengzhou University, Zhengzhou, China.
Wei WangCollege of Public Health, Zhengzhou University, Zhengzhou, China.
Muhammad NaveedDepartment of Physiology and Pharmacology, College of Medicine and Life Sciences, University of Toledo, Toledo, OH, United States.
Qiao ZhangCollege of Public Health, Zhengzhou University, Zhengzhou, China.
Zhengzhou University · CNQuaid-e-Awam University of Engineering, Science and Technology · PKShah Abdul Latif University · PKUniversität Innsbruck · ATUniversity of Toledo · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Cardiovascular disease (CVD) has no socioeconomic, topographical, or sex limitations as reported by the World Health Organization (WHO). The significant drivers of CVD are cardio-metabolic, behavioral, environmental, and social risk factors. However, some significant risk factors for CVD (e.g., a pitiable diet, tobacco smoking, and a lack of physical activities), have also been linked to an elevated risk of cardiovascular disease. Lifestyles and environmental factors are known key variables in cardiovascular disease. The familiarity with smoke goes along with the contact with the environment: air pollution is considered a source of toxins that contribute to the CVD burden. The incidence of myocardial infarction increases in males and females and may lead to fatal coronary artery disease, as confirmed by epidemiological studies. Lipid modification, inflammation, and vasomotor dysfunction are integral components of atherosclerosis development and advancement. These aspects are essential for the identification of atherosclerosis in clinical investigations. This article aims to show the findings on the influence of CVD on the health of individuals and human populations, as well as possible pathology and their involvement in smoking-related cardiovascular diseases. This review also explains lifestyle and environmental factors that are known to contribute to CVD, with indications suggesting an affiliation between cigarette smoking, air pollution, and CVD.

Indexed as

Air PollutionAtherosclerosisCardiovascular DiseasesCigarette SmokingTobacco Smoke PollutionFemaleHumansMaleTobacco Smoke Pollutionair particulate matterair pollutioncardiovascular diseasescigarette smokinginflammation

Identifiers

PMID38045957
PMCPMC10691265
OpenAlexW4388797338

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.