Evidence map›Paper›PMID 37901695›Full record

ReviewGlomerular diseases

Viral Glomerulopathy.

Margaret Deoliveira, Hridyesh Sikri, Samuel Mon-Wei Yu, John Cijiang He

Open access · goldAbstract readReview
In one paragraph

Review in Glomerular diseases. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
0.2field-weighted citation impact, top 46% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 1 citations in OpenAlex.

  1. Review
  2. Post-Transplant Glomerulonephritis: Challenges and Solutions.International journal of nephrology and renovascular disease · 2024
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 1 institution in 1 country.

Margaret DeoliveiraDivision of Nephrology, Department of Medicine, Mount Sinai School of Medicine, New York, NY, USA.
Hridyesh SikriDivision of Nephrology, Department of Medicine, Mount Sinai School of Medicine, New York, NY, USA.
Samuel Mon-Wei YuDivision of Nephrology, Department of Medicine, Mount Sinai School of Medicine, New York, NY, USA.
John Cijiang HeDivision of Nephrology, Department of Medicine, Mount Sinai School of Medicine, New York, NY, USA.
Icahn School of Medicine at Mount Sinai · US

Funding

VIRAL PATHOGENESIS OF HIV ASSOCIATED NEPHROPATHYP01DK056492 · NIDDK · MOUNT SINAI SCHOOL OF MEDICINE OF NYU · PI KLOTMAN, PAUL EVAN · 1999 to 2021
$27.8M
Mount Sinai Nephrology Research Training GrantT32DK007757 · NIDDK · MOUNT SINAI SCHOOL OF MEDICINE OF NYU · PI CAMPBELL, KIRK N, HE, JOHN CIJIANG · 1998 to 2022
$3.8M
Role of RTN1A in the Progression of Diabetic NephropathyR01DK109683 · NIDDK · ICAHN SCHOOL OF MEDICINE AT MOUNT SINAI · PI HE, JOHN CIJIANG · 2016 to 2023
$3.3M
New York Consortium for Interdisciplinary Training in Kidney, Urological and Hematological Research (NYC Train KUHR)U2CDK129502 · NIDDK · ALBERT EINSTEIN COLLEGE OF MEDICINE · PI JONATHAN M. BARASCH, Margaret H Baron · 2022 to 2026
$3.1M
Mechanisms mediating podocyte-parietal epithelial cell crosstalk in proliferative glomerulopathiesR01DK121846 · NIDDK · STATE UNIVERSITY NEW YORK STONY BROOK · PI HE, JOHN CIJIANG, MALLIPATTU, SANDEEP K · 2020 to 2024
$2.7M
Role of RARRES1 in diabetic kidney diseaseR01DK129467 · NIDDK · ICAHN SCHOOL OF MEDICINE AT MOUNT SINAI · PI HE, JOHN CIJIANG, LEE, KYUNG · 2021 to 2024
$1.8M
Role of Protein S in early Diabetic Kidney DiseaseI01BX000345 · VA · JAMES J PETERS VA MEDICAL CENTER · PI HE, JOHN CIJIANG · 2009 to 2024
–
BLRD VA I01 BX000345NIDDK NIH HHS P01 DK056492NIDDK NIH HHS R01 DK109683NIDDK NIH HHS R01 DK121846NIDDK NIH HHS R01 DK129467NIDDK NIH HHS T32 DK007757NIDDK NIH HHS U2C DK129502
6 · The paper itself

Abstract

Background: The association between viral infections and glomerular diseases, commonly known as "viral glomerulopathies," has been described in various clinical scenarios for decades. Despite advancements in diagnostic tools, it remains challenging to establish a causative link fully. Summary: Data from mouse models have substantiated clinical observations and implicate direct viral infection in the pathogenesis of viral glomerulopathy, particularly in human immunodeficiency virus-associated nephropathy. In addition to the traditional concept of direct viral effects on kidneys, other factors such as Key Message: Efforts focusing on vaccines and antiviral treatments have significantly reduced the incidence of viral glomerulopathies. However, the most recent pandemic caused by severe acute respiratory syndrome coronavirus 2 infection complicated by COVID-associated nephropathy illustrates our susceptibility to novel viruses. Ongoing research is pivotal to deciphering the mechanisms behind viral glomerulopathies and discovering therapeutics in a collaborative approach.

Indexed as

COVID-19Hepatitis B virusHepatitis C virusHuman immunodeficiency virusViral glomerulopathy

Identifiers

PMID37901695
PMCPMC10601964
OpenAlexW4384940752

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.