Evidence map›Paper›PMID 37873193›Full record

ArticlebioRxiv : the preprint server for biology2024

Premature endocycling of

Hunter C Herriage, Brian R Calvi

Open access · greenAbstract readPreprint
In one paragraph

Article in bioRxiv : the preprint server for biology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed, 1 citations in OpenAlex.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

2 authors at 2 institutions in 1 country.

Hunter C HerriageDepartment of Biology, Indiana University, Bloomington, IN 47405.ORCID 0000-0003-0127-4022
Brian R CalviDepartment of Biology, Indiana University, Bloomington, IN 47405.ORCID 0000-0001-5304-0047
Indiana University Bloomington · USIndiana University Health · US

Funding

Polypoid cell cycle regulation and genome instabilityR01GM113107 · NIGMS · TRUSTEES OF INDIANA UNIVERSITY · PI CALVI, BRIAN R · 2015 to 2022
$2.6M
NIGMS NIH HHS R01 GM113107
6 · The paper itself

Abstract

Endocycling cells grow and repeatedly duplicate their genome without dividing. Cells switch from mitotic cycles to endocycles in response to developmental signals during the growth of specific tissues in a wide range of organisms. The purpose of switching to endocycles, however, remains unclear in many tissues. Additionally, cells can switch to endocycles in response to conditional signals, which can have beneficial or pathological effects on tissues. However, the impact of these unscheduled endocycles on development is underexplored. Here, we use

Indexed as

border cellsDrosophilaendocyclingepitheliumoogenesispolyploidy

Identifiers

PMID37873193
PMCPMC10592765
OpenAlexW4387532417

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.