Evidence map›Paper›PMID 37867501›Full record

ArticleCanadian journal of kidney health and disease2023

Atypical ADPKD Due to a

Jessica Kachmar, Zaki El-Haffaf, Guillaume Bollée

Open access · goldAbstract readCase Reports
In one paragraph

Article in Canadian journal of kidney health and disease, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
0.6field-weighted citation impact, top 23% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 2 citations in OpenAlex.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Jessica KachmarDivision of Nephrology, Department of Medicine, Centre Hospitalier de l'Université de Montréal, QC, Canada.ORCID https://orcid.org/0009-0007-0543-0025
Zaki El-HaffafDepartment of Genetics, Centre Hospitalier de l'Université de Montréal, QC, Canada.
Guillaume BolléeDivision of Nephrology, Department of Medicine, Centre Hospitalier de l'Université de Montréal, QC, Canada.
Centre Hospitalier de l’Université de Montréal · CA

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Rationale: Due to next-generation sequencing, variants in new genes such as Presenting Concerns of the Patient: We describe a 55-year-old female patient of Haitian origin who presented with slowly deteriorating kidney function, microscopic hematuria, proteinuria, enlarged kidneys with innumerable small cysts, and a family history of chronic kidney disease and cysts. The phenotype was atypical for ADPKD caused by Diagnoses: A panel of genes was analyzed by next-generation massive sequencing techniques, including Interventions: She was treated with candesartan 16 mg once daily to address her proteinuria. Outcomes: At the time of the most recent follow-up, her proteinuria has increased, and her kidney function continues to slowly deteriorate. Teaching Points:

Indexed as

ADPKDchronic kidney diseasecystic kidney diseaseDNAJB11genetic kidney disease

Identifiers

PMID37867501
PMCPMC10585986
OpenAlexW4387756462

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.