Article in EMBO molecular medicine, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
0.7field-weighted citation impact, top 27% of its field
1 · What the graph read from it
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registry
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
3 · Its place in the literature
Who cites it
1 citing paper in PubMed, 4 citations in OpenAlex.
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what money
Authors and funding
13 authors at 4 institutions in 2 countries.
Maria Lourdes MacalinaoSchool of Tropical Medicine and Global Health, Nagasaki University, Nagasaki, Japan.ORCID 0000-0001-7013-9188
Shin-Ichi InoueDivision of Immunology, Department of Molecular Microbiology and Immunology, Graduate School of Biomedical Sciences, Nagasaki University, Nagasaki, Japan.
Sanjaadorj TsogtsaikhanDivision of Immunology, Department of Molecular Microbiology and Immunology, Graduate School of Biomedical Sciences, Nagasaki University, Nagasaki, Japan.
Hirotaka MatsumotoSchool of Information and Data Sciences, Nagasaki University, Nagasaki, Japan.
Ganchimeg BayarsaikhanDivision of Immunology, Department of Molecular Microbiology and Immunology, Graduate School of Biomedical Sciences, Nagasaki University, Nagasaki, Japan.
Jiun-Yu JianDivision of Immunology, Department of Molecular Microbiology and Immunology, Graduate School of Biomedical Sciences, Nagasaki University, Nagasaki, Japan.ORCID 0000-0002-5767-8803
Kazumi KimuraDivision of Immunology, Department of Molecular Microbiology and Immunology, Graduate School of Biomedical Sciences, Nagasaki University, Nagasaki, Japan.
Yoshiaki YasumizuDepartment of Experimental Immunology, Immunology Frontier Research Center, Osaka University, Suita, Japan.ORCID 0000-0002-9872-4909
Tsuyoshi InoueDepartment of Physiology of Visceral Function and Body Fluid, Graduate School of Biomedical Sciences, Nagasaki University, Nagasaki, Japan.
Hiroki YoshidaDivision of Molecular and Cellular Immunoscience, Department of Biomolecular Sciences, Faculty of Medicine, Saga University, Saga, Japan.
Julius HafallaDepartment of Infection Biology, Faculty of Infectious and Tropical Diseases, London School of Hygiene and Tropical Medicine, London, UK.ORCID 0000-0002-5452-9263
Daisuke KimuraDivision of Immunology, Department of Molecular Microbiology and Immunology, Graduate School of Biomedical Sciences, Nagasaki University, Nagasaki, Japan.
Katsuyuki YuiSchool of Tropical Medicine and Global Health, Nagasaki University, Nagasaki, Japan.ORCID 0000-0003-1130-2496
Nagasaki University · JPLondon School of Hygiene & Tropical Medicine · GBSaga University · JPThe University of Osaka · JP
Funding
MEXT | Japan Society for the Promotion of Science (JSPS) 19H03460MEXT | Japan Society for the Promotion of Science (JSPS) 19KK0207MEXT | Japan Society for the Promotion of Science (JSPS) 22H02860MEXT | Japan Society for the Promotion of Science (JSPS) S23038Nagasaki University "Doctoral Program for World-leading Innovative and Smart Education" for Global Health, "Global Health Elite Programme for Building a Healthier World"
6 · The paper itself
Abstract
Malaria infection elicits both protective and pathogenic immune responses, and IL-27 is a critical cytokine that regulate effector responses during infection. Here, we identified a critical window of CD4
Indexed as
Interleukin-27MalariaPlasmodium chabaudiAnimalsCD4-Positive T-LymphocytesMiceMice, Inbred C57BLT-LymphocytesInterleukin-27CD4+ T cellsIL-27immunological memorymalariaTh1
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.
IL-27 produced during acute malaria infection regulates Plasmodium-specific memory CD4 · full record | OpenQuestion