Evidence map›Paper›PMID 37819517›Full record

ArticleMethods in molecular biology (Clifton, N.J.)2024

The Pseudomonas aeruginosa Resistome: Permanent and Transient Antibiotic Resistance, an Overview.

Fernando Sanz-García, Pablo Laborda, Luz Edith Ochoa-Sánchez, José Luis Martínez, Sara Hernando-Amado

Abstract read
PubMed Publisher
In one paragraph

Article in Methods in molecular biology (Clifton, N.J.), 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
1.0field-weighted citation impact, top 25% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 2 citations in OpenAlex.

  1. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 1 institution in 1 country.

Fernando Sanz-GarcíaCentro Nacional de Biotecnología, CSIC, Madrid, Spain.
Pablo LabordaCentro Nacional de Biotecnología, CSIC, Madrid, Spain.
Luz Edith Ochoa-SánchezCentro Nacional de Biotecnología, CSIC, Madrid, Spain.
José Luis MartínezCentro Nacional de Biotecnología, CSIC, Madrid, Spain. jlmtnez@cnb.csic.es.
Sara Hernando-AmadoCentro Nacional de Biotecnología, CSIC, Madrid, Spain.
Centro Nacional de Biotecnología · ES

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

One of the most concerning characteristics of Pseudomonas aeruginosa is its low susceptibility to several antibiotics of common use in clinics, as well as its facility to acquire increased resistance levels. Consequently, the study of the antibiotic resistance mechanisms of this bacterium is of relevance for human health. For such a study, different types of resistance should be distinguished. The intrinsic resistome is composed of a set of genes, present in the core genome of P. aeruginosa, which contributes to its characteristic, species-specific, phenotype of susceptibility to antibiotics. Acquired resistance refers to those genetic events, such as the acquisition of mutations or antibiotic resistance genes that reduce antibiotic susceptibility. Finally, antibiotic resistance can be transiently acquired in the presence of specific compounds or under some growing conditions. The current article provides information on methods currently used to analyze intrinsic, mutation-driven, and transient antibiotic resistance in P. aeruginosa.

Indexed as

Pseudomonas aeruginosaPseudomonas InfectionsAnti-Bacterial AgentsDrug Resistance, MicrobialHumansMicrobial Sensitivity TestsMutationPhenotypeAnti-Bacterial AgentsAcquired resistanceAntibiotic resistanceIntrinsic resistancePseudomonas aeruginosaResistome

Identifiers

PMID37819517
OpenAlexW4387518162

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.