Article in bioRxiv : the preprint server for biology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registry
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
3 · Its place in the literature
Who cites it
1 citing paper in PubMed, 1 citations in OpenAlex.
Seungmae SeoDivision of Allergy, Immunology and Rheumatology, Department of Pediatrics, Columbia University Vagelos College of Physicians and Surgeons; New York, NY, USA, 10032.ORCID 0000-0003-2175-6440
Yong-Oon AhnDivision of Allergy, Immunology and Rheumatology, Department of Pediatrics, Columbia University Vagelos College of Physicians and Surgeons; New York, NY, USA, 10032.ORCID 0000-0001-9904-9581
Sagar L PatilDivision of Allergy, Immunology and Rheumatology, Department of Pediatrics, Columbia University Vagelos College of Physicians and Surgeons; New York, NY, USA, 10032.
Jacqueline ArmettaDivision of Allergy, Immunology and Rheumatology, Department of Pediatrics, Columbia University Vagelos College of Physicians and Surgeons; New York, NY, USA, 10032.
Madrikha D SaturneDivision of Allergy, Immunology and Rheumatology, Department of Pediatrics, Columbia University Vagelos College of Physicians and Surgeons; New York, NY, USA, 10032.
Everardo Hegewisch-SolloaDivision of Allergy, Immunology and Rheumatology, Department of Pediatrics, Columbia University Vagelos College of Physicians and Surgeons; New York, NY, USA, 10032.ORCID 0000-0002-0223-3516
Nicole C GuilzDivision of Allergy, Immunology and Rheumatology, Department of Pediatrics, Columbia University Vagelos College of Physicians and Surgeons; New York, NY, USA, 10032.ORCID 0000-0002-6930-4577
Achchhe PatelColumbia Stem Cell Initiative, Columbia University Irving Medical Center; New York, NY, USA, 10032.
Barbara CorneoColumbia Stem Cell Initiative, Columbia University Irving Medical Center; New York, NY, USA, 10032.ORCID 0000-0001-5189-6749
Malgorzata BorowiakInstitute of Molecular Biology and Biotechnology, Adam Mickiewicz University; Poznan, Poland.ORCID 0000-0002-5829-1213
Emily M MaceDivision of Allergy, Immunology and Rheumatology, Department of Pediatrics, Columbia University Vagelos College of Physicians and Surgeons; New York, NY, USA, 10032.ORCID 0000-0003-0226-7393
Columbia University · USColumbia University Irving Medical Center · USUniversity of Houston · USAdam Mickiewicz University in Poznań · PLGlaxoSmithKline (United States) · USUniversity of Maryland Eastern Shore · US
Funding
GENETIC, IMMUNOLOGIC AND MECHANISTIC BASIS OF HUMAN NK CELL DEFICIENCYR01AI120989 · NIAID · COLUMBIA UNIVERSITY HEALTH SCIENCES · PI ORANGE, JORDAN SCOTT · 2016 to 2025
$7.7M
DEFINING THE ROLE OF CELL MIGRATION IN HUMAN NK CELL DIFFERENTIATIONR01AI137073 · NIAID · COLUMBIA UNIVERSITY HEALTH SCIENCES · PI MACE, EMILY MARGARET · 2018 to 2022
$2.0M
DETERMINING THE ROLE OF THE REPLICATIVE HELICASE IN HUMAN NK CELL DEVELOPMENTR01AI137275 · NIAID · COLUMBIA UNIVERSITY HEALTH SCIENCES · PI MACE, EMILY MARGARET · 2018 to 2022
While monogenic variants in CDC45-MCM-GINS (CMG) replisome proteins cause human natural killer cell deficiencies (NKD), family members with the same inherited variants often have variable clinical and cellular phenotypes. We investigated two siblings with inherited compound heterozygous
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.
Allelic bias contributes to heterogeneous phenotypes of NK cell deficiency. · full record | OpenQuestion