Evidence map›Paper›PMID 37762342›Full record

ArticleInternational journal of molecular sciences2023

The Role of

Elisabetta Bolognesi, Franca Rosa Guerini, Alessandra Carta, Matteo Chiappedi, Stefano Sotgiu, Martina Maria Mensi, Cristina Agliardi, Milena Zanzottera, Mario Clerici

Open access · goldAbstract read
In one paragraph

Article in International journal of molecular sciences, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
1.2field-weighted citation impact, top 22% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 7 citations in OpenAlex.

  1. Review
  2. Article
  3. Decoding the genetic landscape of autism: A comprehensive review.World journal of clinical pediatrics · 2024
    Article
  4. Editorial for theInternational journal of molecular sciences · 2024
    Article
  5. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 3 institutions in 1 country.

Elisabetta BolognesiLaboratory of Molecular Medicine and Biotechnology, IRCCS Fondazione Don Carlo Gnocchi, Via Capecelatro 66, 20148 Milan, Italy.ORCID 0000-0001-5554-1697
Franca Rosa GueriniLaboratory of Molecular Medicine and Biotechnology, IRCCS Fondazione Don Carlo Gnocchi, Via Capecelatro 66, 20148 Milan, Italy.ORCID 0000-0001-9461-5927
Alessandra CartaUnit of Child Neuropsychiatry, Department of Medicine, Surgery and Pharmacy, University of Sassari, 07100 Sassari, Italy.
Matteo ChiappediChild Neuropsychiatry Unit, IRCCS Mondino Foundation, 27100 Pavia, Italy.ORCID 0000-0002-2682-5006
Stefano SotgiuUnit of Child Neuropsychiatry, Department of Medicine, Surgery and Pharmacy, University of Sassari, 07100 Sassari, Italy.
Martina Maria MensiChild Neuropsychiatry Unit, IRCCS Mondino Foundation, 27100 Pavia, Italy.ORCID 0000-0002-9583-0554
Cristina AgliardiLaboratory of Molecular Medicine and Biotechnology, IRCCS Fondazione Don Carlo Gnocchi, Via Capecelatro 66, 20148 Milan, Italy.ORCID 0000-0003-2022-3386
Milena ZanzotteraLaboratory of Molecular Medicine and Biotechnology, IRCCS Fondazione Don Carlo Gnocchi, Via Capecelatro 66, 20148 Milan, Italy.
Mario ClericiLaboratory of Molecular Medicine and Biotechnology, IRCCS Fondazione Don Carlo Gnocchi, Via Capecelatro 66, 20148 Milan, Italy.ORCID 0000-0001-5920-6191
Don Carlo Gnocchi Foundation · ITUniversity of Sassari · ITUniversity of Milan · IT

Funding

Fondazione Alessandro and Vincenzo Negroni Prati MorosiniFondazione Romeo and Enrica Invernizzi.Italian Ministry of Health Ricerca Corrente 2022
6 · The paper itself

Abstract

Autism spectrum disorders (ASD) can present with different onset and timing of symptom development; children may manifest symptoms early in their first year of life, i.e., early onset (EO-ASD), or may lose already achieved skills during their second year of life, thus showing a regressive-type onset (RO-ASD). It is still controversial whether regression represents a neurobiological subtype of ASD, resulting from distinct genetic and environmental causes. We focused this study on the 25 kD synaptosomal-associated protein (

Indexed as

Autism Spectrum DisorderAutistic DisorderMicroRNAs3' Untranslated RegionsAllelesChildGenotypeHumansSynaptosomal-Associated Protein 253' Untranslated RegionsMicroRNAsSNAP25 protein, humanSynaptosomal-Associated Protein 25ASD classic onsetASD regressive onsetautism spectrum disorders (ASD)epigeneticsregressionSNAP-25 gene (synaptosomal-associated protein of 25 kDa)

Identifiers

PMID37762342
PMCPMC10531097
OpenAlexW4386749140

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.