Evidence map›Paper›PMID 37762211›Full record

ReviewInternational journal of molecular sciences2023

The Exon 3-Deleted Growth Hormone Receptor (d3GHR) Polymorphism-A Favorable Backdoor Mechanism for the GHR Function.

Ghadeer Falah, Lital Sharvit, Gil Atzmon

Open access · goldAbstract readReview
In one paragraph

Review in International journal of molecular sciences, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
0.6field-weighted citation impact, top 28% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 3 citations in OpenAlex.

  1. Review
  2. Review
  3. Article
  4. Article
  5. Observational
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 2 institutions in 2 countries.

Ghadeer FalahFaculty of Natural Sciences, University of Haifa, Haifa 3498838, Israel.ORCID 0009-0003-7151-0024
Lital SharvitFaculty of Natural Sciences, University of Haifa, Haifa 3498838, Israel.ORCID 0000-0002-2577-1907
Gil AtzmonFaculty of Natural Sciences, University of Haifa, Haifa 3498838, Israel.ORCID 0000-0001-6895-3607
University of Haifa · ILAlbert Einstein College of Medicine · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Growth hormone (GH) is a peptide hormone that plays a crucial role in controlling growth, development, and lifespan. Molecular regulation of GH is accomplished via the

Indexed as

ExonsPolymorphism, GeneticReceptors, SomatotropinAcromegalyAnimalsHuman Growth HormoneHumansInsulin-Like Growth Factor IPrader-Willi SyndromeProtein IsoformsHuman Growth HormoneInsulin-Like Growth Factor IProtein IsoformsReceptors, Somatotropindeletion of exon 3growth and developmentgrowth hormone receptorhormone deficiencyhuman growth hormonepolymorphism

Identifiers

PMID37762211
PMCPMC10531306
OpenAlexW4386616797

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.