Evidence map›Paper›PMID 37723445›Full record

ArticleCellular & molecular biology letters2023

S100a9 inhibits Atg9a transcription and participates in suppression of autophagy in cardiomyocytes induced by β

Xiaoyan Zhi, Shu Shi, Yang Li, Mingxia Ma, Yaolin Long, Chen Li, Haihu Hao, Huirong Liu, Xiaohui Wang, Li Wang

Open access · goldAbstract read
In one paragraph

Article in Cellular & molecular biology letters, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
2.1field-weighted citation impact, top 12% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 10 citations in OpenAlex.

  1. Article
  2. Article
  3. Progress on multifunctional transmembrane protein ATG9A.Cell communication and signaling : CCS · 2025
    Review
  4. Article
  5. Quercetin reverses βFrontiers in nutrition · 2025
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 2 institutions in 1 country.

Xiaoyan ZhiDepartment of Pathology, Shanxi Medical University, No.56 Xinjian South Road, Taiyuan, Shanxi, 030001, People's Republic of China.
Shu ShiDepartment of Pathology, Shanxi Medical University, No.56 Xinjian South Road, Taiyuan, Shanxi, 030001, People's Republic of China.
Yang LiDepartment of Pathology, Shanxi Medical University, No.56 Xinjian South Road, Taiyuan, Shanxi, 030001, People's Republic of China.
Mingxia MaDepartment of Pathology, Shanxi Medical University, No.56 Xinjian South Road, Taiyuan, Shanxi, 030001, People's Republic of China.
Yaolin LongDepartment of Pathology, Shanxi Medical University, No.56 Xinjian South Road, Taiyuan, Shanxi, 030001, People's Republic of China.
Chen LiDepartment of Pathology, Shanxi Medical University, No.56 Xinjian South Road, Taiyuan, Shanxi, 030001, People's Republic of China.
Haihu HaoDepartment of Orthopaedics, Shanxi Bethune Hospital, Shanxi Academy of Medical Science, Tongji Shanxi Hospital, Third Hospital of Shanxi Medical University, Taiyuan, People's Republic of China.
Huirong LiuDepartment of Physiology and Pathophysiology, School of Basic Medical Sciences, Capital Medical University, Beijing, People's Republic of China.
Xiaohui WangDepartment of Pathology, Shanxi Medical University, No.56 Xinjian South Road, Taiyuan, Shanxi, 030001, People's Republic of China.
Li WangDepartment of Pathology, Shanxi Medical University, No.56 Xinjian South Road, Taiyuan, Shanxi, 030001, People's Republic of China. mirror0117@126.com.ORCID http://orcid.org/0000-0002-4206-0285
Shanxi Medical University · CNCapital Medical University · CN

Funding

Basic Research Project of the Shanxi Science and Technology Department No.202203021222257Cultivate Scientific Research Excellence Programs of Higher Education Institutions in Shanxi No.2020KJ034National Natural Science Foundation of China 31871177National Natural Science Foundation of China 82271523the basic research project of Shanxi Science and Technology Department No.202103021224347
6 · The paper itself

Abstract

backgroundCardiomyocyte death induced by autophagy inhibition is an important cause of cardiac dysfunction. In-depth exploration of its mechanism may help to improve cardiac dysfunction. In our previous study, we found that β

methodsIn this study, the active immunity method was used to establish a β

resultsThe results showed that β

conclusionOur research showed that β

Indexed as

Calgranulin BMyocytes, CardiacAnimalsAutoantibodiesAutophagyAutophagy-Related ProteinsDisease Models, AnimalMembrane ProteinsMiceMyocardiumVesicular Transport ProteinsAtg9A protein, mouseAutoantibodiesAutophagy-Related ProteinsCalgranulin BMembrane ProteinsS100A9 protein, mouseVesicular Transport ProteinsAtg9aAutophagyHIF-1αS100a9β1-AA

Identifiers

PMID37723445
PMCPMC10506287
OpenAlexW4386837534

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.