Evidence map›Paper›PMID 37692837›Full record

ArticleJournal of immunology research2023

Effect of Differently Polarized Human Macrophages on the SH-SY5Y Cells Damaged by Ischemia and Hypoxia

Ivan Mikhailovich Rashchupkin, Ekaterina Yakovlevna Shevela, Aleksandra Aleksandrovna Maksimova, Marina Aleksandrovna Tikhonova, Aleksandr Anatolevich Ostanin, Elena Removna Chernykh

Open access · goldAbstract read
In one paragraph

Article in Journal of immunology research, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
0.3field-weighted citation impact, top 34% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 2 citations in OpenAlex.

  1. Article
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 1 institution in 1 country.

Ivan Mikhailovich RashchupkinScientific Research Institute of Fundamental and Clinical Immunology, Novosibirsk, Russia.ORCID https://orcid.org/0009-0007-3528-3783
Ekaterina Yakovlevna ShevelaScientific Research Institute of Fundamental and Clinical Immunology, Novosibirsk, Russia.ORCID https://orcid.org/0000-0001-8997-3586
Aleksandra Aleksandrovna MaksimovaScientific Research Institute of Fundamental and Clinical Immunology, Novosibirsk, Russia.ORCID https://orcid.org/0000-0003-0884-0226
Marina Aleksandrovna TikhonovaScientific Research Institute of Fundamental and Clinical Immunology, Novosibirsk, Russia.ORCID https://orcid.org/0000-0002-2366-1667
Aleksandr Anatolevich OstaninScientific Research Institute of Fundamental and Clinical Immunology, Novosibirsk, Russia.ORCID https://orcid.org/0000-0001-6895-938X
Elena Removna ChernykhScientific Research Institute of Fundamental and Clinical Immunology, Novosibirsk, Russia.ORCID https://orcid.org/0000-0003-2346-6279
Research Institute of Fundamental and Clinical Immunology · RU

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Macrophages are the major cells of innate immunity with a wide range of biological effects due to their great plasticity and heterogeneity. Macrophages play a key role in neuroregeneration following nervous tissue injury. However, the neuroregenerative potential of various macrophage phenotypes, including those polarized by efferocytosis, remains unexplored. The aim of this study was to compare the neuroregenerative and neuroprotective activity of soluble factors secreted by variously activated human macrophages on the functions of neural progenitors in an

Indexed as

NeuroblastomaHumansHypoxiaIschemiaMacrophagesPhagocytosis

Identifiers

PMID37692837
PMCPMC10484653
OpenAlexW4386307467

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.