Evidence map›Paper›PMID 37663801›Full record

ArticleToxicology research2023

Investigation of oxidative stress status and apoptotic markers of juvenile trout exposed to arsenic toxicity.

Hasan Saç, Aslı Ç Yeltekin

Open access · greenAbstract read
In one paragraph

Article in Toxicology research, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
1.1field-weighted citation impact, top 25% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 8 citations in OpenAlex.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 1 institution in 1 country.

Hasan SaçDepartment of Chemistry, Faculty of Science, University of Yüzüncü Yıl, Van 65040, Turkey.
Aslı Ç YeltekinDepartment of Chemistry, Faculty of Science, University of Yüzüncü Yıl, Van 65040, Turkey.ORCID https://orcid.org/0000-0003-0071-7434
Van Yüzüncü Yıl Üniversitesi · TR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Arsenic is one of the most abundant elements on earth. Arsenic, also called metalloid, is used as a raw material in many industries. Arsenic causes the acceleration of free radical production in the body and the resulting oxidative stress. In juvenile trout, the interactions of arsenic with metacomposition, biochemical analysis, and apoptosis stimuli were investigated. Results were demonstrated by several marker applications, including oxidative stress parameters, proinflammatory cytokine expressions, DNA damage, and apoptosis markers. In our study, arsenic was applied to juvenile trout (

Indexed as

arseniccytokinesNF-kBNrf-2rainbow trout

Identifiers

PMID37663801
PMCPMC10470348
OpenAlexW4383033132

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.