Evidence map›Paper›PMID 37628604›Full record

ArticleGenes2023

Arabidopsis RAD16 Homologues Are Involved in UV Tolerance and Growth.

Linda Alrayes, Jake Stout, Dana Schroeder

Open access · goldAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Genes, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
2.0field-weighted citation impact, top 12% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 5 citations in OpenAlex.

  1. Plant tolerance mechanisms to DNA-damaging UV stress.Journal of experimental botany · 2025
    Review
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Linda AlrayesDepartment of Biological Sciences, University of Manitoba, Winnipeg, MB R3T 2N2, Canada.
Jake StoutDepartment of Biological Sciences, University of Manitoba, Winnipeg, MB R3T 2N2, Canada.ORCID 0000-0002-0193-9066
Dana SchroederDepartment of Biological Sciences, University of Manitoba, Winnipeg, MB R3T 2N2, Canada.
University of Manitoba · CA

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

In plants, prolonged exposure to ultraviolet (UV) radiation causes harmful DNA lesions. Nucleotide excision repair (NER) is an important DNA repair mechanism that operates via two pathways: transcription coupled repair (TC-NER) and global genomic repair (GG-NER). In plants and mammals, TC-NER is initiated by the Cockayne Syndrome A and B (CSA/CSB) complex, whereas GG-NER is initiated by the Damaged DNA Binding protein 1/2 (DDB1/2) complex. In the yeast

Indexed as

ArabidopsisCockayne SyndromeSaccharomyces cerevisiae ProteinsAdenosine TriphosphatasesAnimalsDNA-Binding ProteinsDNA RepairMammalsSaccharomyces cerevisiaeUltraviolet RaysAdenosine TriphosphatasesDNA-Binding ProteinsRAD16 protein, S cerevisiaeRAD7 protein, S cerevisiaeSaccharomyces cerevisiae ProteinsArabidopsisCSA/CSBDDB1/2Nucleotide excision repairRAD7/16UV sensitivity

Identifiers

PMID37628604
PMCPMC10454142
OpenAlexW4385342157

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.