Evidence map›Paper›PMID 37593235›Full record

ArticleFrontiers in physiology2023

Lineage-specific mutation of

Angel Cabrera Pereira, Krishnakali Dasgupta, Thach-Vu Ho, Maria Pacheco-Vergara, Julie Kim, Niam Kataria, Yaowei Liang, Jeslyn Mei, Jinyeong Yu, Lukasz Witek and 2 more

Open access · goldAbstract read
In one paragraph

Article in Frontiers in physiology, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
0.2field-weighted citation impact, top 41% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed, 1 citations in OpenAlex.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors at 3 institutions in 1 country.

Angel Cabrera PereiraDepartment of Molecular Pathobiology, College of Dentistry, New York University, New York, NY, United States.
Krishnakali DasguptaDepartment of Molecular Pathobiology, College of Dentistry, New York University, New York, NY, United States.
Thach-Vu HoCenter for Craniofacial Molecular Biology, University of Southern California, Los Angeles, CA, United States.
Maria Pacheco-VergaraDepartment of Molecular Pathobiology, College of Dentistry, New York University, New York, NY, United States.
Julie KimDepartment of Molecular Pathobiology, College of Dentistry, New York University, New York, NY, United States.
Niam KatariaDepartment of Molecular Pathobiology, College of Dentistry, New York University, New York, NY, United States.
Yaowei LiangDepartment of Molecular Pathobiology, College of Dentistry, New York University, New York, NY, United States.
Jeslyn MeiDepartment of Molecular Pathobiology, College of Dentistry, New York University, New York, NY, United States.
Jinyeong YuDepartment of Molecular Pathobiology, College of Dentistry, New York University, New York, NY, United States.
Lukasz WitekBiomaterials Division, New York University College of Dentistry, New York, NY, United States.
Yang ChaiCenter for Craniofacial Molecular Biology, University of Southern California, Los Angeles, CA, United States.
Juhee JeongDepartment of Molecular Pathobiology, College of Dentistry, New York University, New York, NY, United States.
New York University · USUniversity of Southern California · USFlorida State University · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The calvaria (top part of the skull) is made of pieces of bone as well as multiple soft tissue joints called sutures. The latter is crucial to the growth and morphogenesis of the skull, and thus a loss of calvarial sutures can lead to severe congenital defects in humans. During embryogenesis, the calvaria develops from the cranial mesenchyme covering the brain, which contains cells originating from the neural crest and the mesoderm. While the mechanism that patterns the cranial mesenchyme into bone and sutures is not well understood, function of

Indexed as

calvariacraniofacial developmentcraniosynostosisembryoLMX1Bmouse

Identifiers

PMID37593235
PMCPMC10427921
OpenAlexW4385455864

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.