Evidence map›Paper›PMID 37542675›Full record

ArticleJournal of neural transmission (Vienna, Austria : 1996)2023

Molecular signature of excessive female aggression: study of stressed mice with genetic inactivation of neuronal serotonin synthesis.

Tatyana Strekalova, Oleg Moskvin, Aayushi Y Jain, Nikita Gorbunov, Anna Gorlova, Daria Sadovnik, Aleksei Umriukhin, Raymond Cespuglio, Wing Shan Yu, Anna Chung Kwan Tse and 3 more

Open access · hybridAbstract read
In one paragraph

Article in Journal of neural transmission (Vienna, Austria : 1996), 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
2.0field-weighted citation impact, top 14% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed, 6 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors at 7 institutions in 7 countries.

Tatyana Strekalova *Division of Molecular Psychiatry, Center of Mental Health, University Hospital of Würzburg, Würzburg, Germany.
Oleg Moskvin *Primate Research Center, University of Wisconsin-Madison, Madison, WI, USA.
Aayushi Y Jain *Department of Biomolecular Chemistry, School of Medicine and Public Health, University of Wisconsin-Madison, Madison, WI, USA.
Nikita Gorbunov *Division of Molecular Psychiatry, Center of Mental Health, University Hospital of Würzburg, Würzburg, Germany.
Anna GorlovaLaboratory of Psychiatric Neurobiology, Institute of Molecular Medicine and Department of Normal Physiology, Sechenov Moscow State Medical University, Moscow, Russia.
Daria SadovnikLaboratory of Psychiatric Neurobiology, Institute of Molecular Medicine and Department of Normal Physiology, Sechenov Moscow State Medical University, Moscow, Russia.
Aleksei UmriukhinLaboratory of Psychiatric Neurobiology, Institute of Molecular Medicine and Department of Normal Physiology, Sechenov Moscow State Medical University, Moscow, Russia.
Raymond CespuglioLaboratory of Psychiatric Neurobiology, Institute of Molecular Medicine and Department of Normal Physiology, Sechenov Moscow State Medical University, Moscow, Russia.
Wing Shan YuNeuromodulation Laboratory, School of Biomedical Sciences, Li Ka Shing Faculty of Medicine, The University of Hong Kong, Pok Fu Lam, Hong Kong SAR, China.
Anna Chung Kwan TseNeuromodulation Laboratory, School of Biomedical Sciences, Li Ka Shing Faculty of Medicine, The University of Hong Kong, Pok Fu Lam, Hong Kong SAR, China.
Allan V KalueffInstitute of Translational Biomedicine, St. Petersburg State University, St. Petersburg, Russia.
Klaus-Peter LeschDivision of Molecular Psychiatry, Center of Mental Health, University Hospital of Würzburg, Würzburg, Germany. kplesch@mail.uni-wuerzburg.de.
Lee Wei LimNeuromodulation Laboratory, School of Biomedical Sciences, Li Ka Shing Faculty of Medicine, The University of Hong Kong, Pok Fu Lam, Hong Kong SAR, China. drlimleewei@gmail.com.
Sechenov University · RUUniversity of Hong Kong · HKMaastricht University · NLInstitute of Medical Biology · SGSt Petersburg University · RUUniversitätsklinikum Würzburg · DEUniversity of Wisconsin–Madison · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Aggression is a complex social behavior, critically involving brain serotonin (5-HT) function. The neurobiology of female aggression remains elusive, while the incidence of its manifestations has been increasing. Yet, animal models of female aggression are scarce. We previously proposed a paradigm of female aggression in the context of gene x environment interaction where mice with partial genetic inactivation of tryptophan hydroxylase-2 (Tph2

Indexed as

SerotoninTryptophan HydroxylaseAggressionAnimalsBrainFemaleMiceRatsSocial BehaviorSerotoninTryptophan HydroxylaseAggressionDeep sequencing (mRNAseq)MicePredation stressPrefrontal cortexSerotoninTryptophan hydroxylase-2 (Tph2)

Identifiers

PMID37542675
PMCPMC10460733
OpenAlexW4385602349

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.