Evidence map›Paper›PMID 37523556›Full record

ArticleProceedings of the National Academy of Sciences of the United States of America2023

SEC61G assists

Kunlin Zeng, Yu Zeng, Hongchao Zhan, Ziling Zhan, Li Wang, Yuxin Xie, Yanqing Tang, Cuiying Li, Yanwen Chen, Shangbiao Li and 6 more

Open access · greenAbstract read
In one paragraph

Article in Proceedings of the National Academy of Sciences of the United States of America, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 25 papers.

0numbers the graph read from it
0cells of the map it votes in
25citing papers in PubMed
7.8field-weighted citation impact, top 2% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

25 citing papers in PubMed, 27 citations in OpenAlex.

  1. Trial
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  13. Glycosylation Gene Signatures as Prognostic Biomarkers in Glioblastoma.Annals of clinical and translational neurology · 2025
    Article
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4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

16 authors at 3 institutions in 1 country.

Kunlin ZengDepartment of Cell Biology, School of Basic Medical Science, Southern Medical University, Guangzhou 510515, China.
Yu ZengDepartment of Cell Biology, School of Basic Medical Science, Southern Medical University, Guangzhou 510515, China.
Hongchao ZhanDepartment of Cell Biology, School of Basic Medical Science, Southern Medical University, Guangzhou 510515, China.
Ziling ZhanDepartment of Cell Biology, School of Basic Medical Science, Southern Medical University, Guangzhou 510515, China.
Li WangDepartment of Cell Biology, School of Basic Medical Science, Southern Medical University, Guangzhou 510515, China.
Yuxin XieDepartment of Cell Biology, School of Basic Medical Science, Southern Medical University, Guangzhou 510515, China.
Yanqing TangDepartment of Cell Biology, School of Basic Medical Science, Southern Medical University, Guangzhou 510515, China.
Cuiying LiDepartment of Cell Biology, School of Basic Medical Science, Southern Medical University, Guangzhou 510515, China.
Yanwen ChenDepartment of Cell Biology, School of Basic Medical Science, Southern Medical University, Guangzhou 510515, China.
Shangbiao LiDepartment of Cell Biology, School of Basic Medical Science, Southern Medical University, Guangzhou 510515, China.ORCID 0000-0002-7536-5552
Ming LiuDepartment of Cell Biology, School of Basic Medical Science, Southern Medical University, Guangzhou 510515, China.ORCID 0000-0003-3567-530X
Xiaoxia ChenDepartment of Cell Biology, School of Basic Medical Science, Southern Medical University, Guangzhou 510515, China.
Li LiangGuangdong Province Key Laboratory of Molecular Tumor Pathology, School of Basic Medical Science, Southern Medical University, Guangzhou 510515, China.
Fan DengDepartment of Cell Biology, School of Basic Medical Science, Southern Medical University, Guangzhou 510515, China.
Ye SongDepartment of Neurosurgery, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
Aidong ZhouDepartment of Cell Biology, School of Basic Medical Science, Southern Medical University, Guangzhou 510515, China.
Southern Medical University · CNZhujiang Hospital · CNNanfang Hospital · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Amplification of chromosome 7p11 (7p11) is the most common alteration in primary glioblastoma (GBM), resulting in gains of epidermal growth factor receptor (

Indexed as

Brain NeoplasmsGlioblastomaSEC Translocation ChannelsAnimalsCD8-Positive T-LymphocytesCell Line, TumorErbB ReceptorsMiceErbB ReceptorsSec61g protein, mouseSEC Translocation ChannelsEGFRglioblastomaimmune evasionPD-L1SEC61G

Identifiers

PMID37523556
PMCPMC10410745
OpenAlexW4385409470

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.