ArticleJCPP advances2022
Subdimensions of social-communication behavior in autism-A replication study.
Article in JCPP advances, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
4 citing papers in PubMed, 6 citations in OpenAlex.
- The Developmental Assessment of Social Communication Ability (DASCA): initial creation and psychometric description.Molecular autism · 2025Article
- Autism spectrum disorder in ICD-11-a critical reflection of its possible impact on clinical practice and research.Molecular psychiatry · 2024Review
- Conceptualization of the latent structure of autism: further evidence and discussion of dimensional and hybrid models.European child & adolescent psychiatry · 2023Article
- Subdimensions of social-communication behavior in autism-A replication study.JCPP advances · 2022Article
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Authors and funding
7 authors at 3 institutions in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Introduction: In order to identify more refined dimensions of social-communication impairments in autism spectrum disorder (ASD) a previous study applied exploratory and confirmatory factor analyses to diagnostic algorithm scores of the autism diagnostic observation schedule (ADOS), Module 3. A three-factor model consisting of repetitive behaviors, impairments in 'Basic Social-Communication' and in 'Interaction quality' (IQ) was established and confirmed. The current study aimed to replicate this model in an independent sample. To advance our understanding of the latent structure of social communication deficits, previous work was complemented by a probabilistic approach. Methods: Participants ( Results: Results confirmed the previously reported observation of three latent dimensions in the ADOS algorithm reflecting 'Restricted, Repetitive Behaviors', 'Basic Social-Communication' behaviors and 'Interaction Quality'. All three dimensions contributed independently and additively to the prediction of an ASD diagnosis. Conclusion: By replicating previous findings in a large clinical sample our results contribute to further conceptualize the social-communication impairments in ASD as two dimensional.
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Registered trials
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