Evidence map›Paper›PMID 37405188›Full record

ArticleJournal of physical therapy science2023

Glucose loading for heart failure protects the myocardium and improves physical function.

Ryoichi Nishida, Jun Goto, Noboru Kamajiri, Shogo Terayama, Mimo Shirahase, Hidetaka Imagita

Open access · diamondAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Journal of physical therapy science, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
0.2field-weighted citation impact, top 48% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 1 citations in OpenAlex.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 5 institutions in 1 country.

Ryoichi NishidaTakanohara Central Hospital: 1-3-3 Ukyo, Nara-shi, Nara 631-0805, Japan.
Jun GotoDepartment of Anatomy and Cell Biology, Graduate School of Medicine, Osaka Metropolitan University, Japan.
Noboru KamajiriKishiwada Rehabilitation Hospital, Japan.
Shogo TerayamaDepartment of Physical Therapy, Faculty of Health Science, Kio University, Japan.
Mimo ShirahaseKishiwada Rehabilitation Hospital, Japan.
Hidetaka ImagitaDepartment of Physical Therapy, Health and Social Services, Saitama Prefectural University, Japan.
Kishiwada City Hospital · JPKio University · JPNara Medical University · JPOsaka Metropolitan UniversitySaitama Prefectural University · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

[Purpose] The purpose of this study was to investigate the effects of glucose intake on physical function in a heart failure rat model. [Materials and Methods] Five-week-old male Wistar rats were used for this study. Monocrotalin (40 mg/kg) was administered intraperitoneally to rats to induce heart failure. The rats were divided into two groups, control and MCT; the MCT group was further classified according to glucose concentration (0%, 10%, and 50%). [Results] Glucose intake during heart failure prevented the loss of body weight, skeletal muscle, and fat mass. Myocardial metabolism in heart failure was enhanced by hypoxia, which in turn, enhanced the glycolytic system. [Conclusion] Glucose loading suppressed cardiac hypertrophy and improved physical function in the heart failure rat model.

Indexed as

Cardiac cachexiaHeart failureMyocardium metabolism

Identifiers

PMID37405188
PMCPMC10315208
OpenAlexW4382652475

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.