Article in Life science alliance, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
0.3field-weighted citation impact, top 37% of its field
1 · What the graph read from it
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registry
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
3 · Its place in the literature
Who cites it
2 citing papers in PubMed, 2 citations in OpenAlex.
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what money
Authors and funding
9 authors at 4 institutions in 3 countries.
Huahua LiuDepartment of Cardiology, First Affiliated Hospital, Xi'an Jiaotong University, Xi'an, China.
Pengfei LuDepartment of Genetics, Albert Einstein College of Medicine, Bronx, NY, USA.
Shan HeThe Institute of Cardiovascular Sciences, School of Basic Medical Sciences, Xi'an Jiaotong University, Xi'an, China.
Yuru LuoThe Institute of Cardiovascular Sciences, School of Basic Medical Sciences, Xi'an Jiaotong University, Xi'an, China.
Yuan FangThe Institute of Cardiovascular Sciences, School of Basic Medical Sciences, Xi'an Jiaotong University, Xi'an, China.
Sonia BenkaciDepartment of Genetics, Albert Einstein College of Medicine, Bronx, NY, USA.
Bingruo WuDepartment of Genetics, Albert Einstein College of Medicine, Bronx, NY, USA.
Yidong WangThe Institute of Cardiovascular Sciences, School of Basic Medical Sciences; Department of Cardiology, First Affiliated Hospital; Key Laboratory of Environment and Genes Related to Diseases of Ministry of Education, Xi'an Jiaotong University, Xi'an, China yidwang119@xjtu.edu.cn.ORCID 0000-0001-7718-4352
Bin ZhouDepartments of Genetics, Pediatrics (Pediatric Genetic Medicine), and Medicine (Cardiology), The Wilf Family Cardiovascular Research Institute, The Einstein Institute for Aging Research, Albert Einstein College of Medicine, Bronx, NY, USA bin.zhou@einsteinmed.edu.ORCID 0000-0003-0832-4002
Albert Einstein College of Medicine · USXi'an Jiaotong University · CNFirst Affiliated Hospital of Xi'an Jiaotong University · CNMinistry of Education
Funding
Molecular and Cellular Mechanisms in Coronary Artery Development and AnomaliesR01HL133120 · NHLBI · UNIVERSITY OF CHICAGO · PI BIN ZHOU · 2016 to 2026
$4.6M
Molecular signaling in aortic valve development and congenital aortic valve defectR01HL157347 · NHLBI · UNIVERSITY OF CHICAGO · PI ZHENG, DEYOU, ZHOU, BIN · 2022 to 2025
$2.6M
Control of cardiomyocyte cell cycle by REST in heart failure and regenerationR01HL148128 · NHLBI · UNIVERSITY OF CHICAGO · PI ZHENG, DEYOU, ZHOU, BIN · 2020 to 2023
$2.5M
Molecular and cellular mechanisms in cardiac outflow tract formation and defectsR01HL159515 · NHLBI · UNIVERSITY OF CHICAGO · PI MORROW, BERNICE E, ZHOU, BIN · 2021 to 2024
Endocardial cushion formation is essential for heart valve development and heart chamber separation. Abnormal endocardial cushion formation often causes congenital heart defects. β-Catenin is known to be essential for endocardial cushion formation; however, the underlying cellular and molecular mechanisms remain incompletely understood. Here, we show that endothelial-specific deletion of β-catenin in mice resulted in formation of hypoplastic endocardial cushions due to reduced cell proliferation and impaired cell migration. By using a
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.
β-Catenin regulates endocardial cushion growth by suppressing p21. · full record | OpenQuestion